J. Bentley

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phenotypic maturation in airway smooth muscle Akt activation induces hypertrophy without contractile

J. McConville, N. Dulin, B. Camoretti-Mercado, 2016 .

Akt activation induces hypertrophy without contractile phenotypic maturation in airway smooth muscle.

J. McConville, N. Dulin, B. Camoretti-Mercado, 2011, American journal of physiology. Lung cellular and molecular physiology.

p70 Ribosomal S6 kinase is required for airway smooth muscle cell size enlargement but not increased contractile protein expression.

D. Fingar, K. Bitar, J. Solway, 2010, American journal of respiratory cell and molecular biology.

Deficient inflammasome activation permits an exaggerated asthma phenotype in rhinovirus C-infected immature mice

Mingyuan Han, M. Hershenson, T. Ishikawa, 2021, Mucosal Immunology.

Transforming growth factor-β induces airway smooth muscle hypertrophy

D. Fingar, K. Bitar, M. Hershenson, 2006 .

Macrophages Release from Functionally Polarized and -Challenged Mice Induces Eotaxin Rhinovirus Infection of Allergen-Sensitized

Ying Zhao, N. Lukacs, C. McHenry, 2010 .

Myristoylated rhinovirus VP4 protein activates TLR2-dependent proinflammatory gene expression.

C. Rajput, Mingyuan Han, J. Lei, 2019, American journal of physiology. Lung cellular and molecular physiology.

Ovalbumin sensitization and challenge increases the number of lung cells possessing a mesenchymal stromal cell phenotype

Amy E. Baek, J. Lei, M. Hershenson, 2010, Respiratory research.

TLR2-expressing macrophages are required and sufficient for rhinovirus-induced airway inflammation

Yutein Chung, C. Rajput, Mingyuan Han, 2016 .

Transforming growth factor-beta induces airway smooth muscle hypertrophy.

D. Fingar, K. Bitar, M. Hershenson, 2006, American journal of respiratory cell and molecular biology.

Airway smooth muscle hyperplasia and hypertrophy correlate with glycogen synthase kinase-3(beta) phosphorylation in a mouse model of asthma.

W. Henderson, D. Fingar, K. Bitar, 2009, American journal of physiology. Lung cellular and molecular physiology.

Airway smooth muscle hyperplasia and hypertrophy correlate with glycogen synthase kinase-3 phosphorylation in a mouse model of asthma

W. Henderson, D. Fingar, K. Bitar, 2009 .

Pellino-1 Regulates the Responses of the Airway to Viral Infection

J. Stewart, Shao-Cong Sun, W. Timens, 2018, Frontiers in Cellular and Infection Microbiology.

Pulmonary artery smooth muscle hypertrophy: roles of glycogen synthase kinase-3beta and p70 ribosomal S6 kinase.

D. Pinsky, A. C. Anyanwu, J. Lei, 2010, American journal of physiology. Lung cellular and molecular physiology.

HyperresponsivenessMucous Metaplasia and Airways Neonatal Rhinovirus Infection Induces

J. Curtis, Ying Zhao, N. Lukacs, 2012 .

Nerve growth factor inhibits PC12 cell PDE 2 phosphodiesterase activity and increases PDE 2 binding to phosphoproteins

D. Juilfs, M. Uhler, J. Bentley, 2001, Journal of neurochemistry.

M2 Macrophages promote IL-33 expression, ILC2 expansion and mucous metaplasia in response to early life rhinovirus infections

Mingyuan Han, M. Hershenson, Yiran Li, 2022, Frontiers in Immunology.

Hyperoxic Exposure of Immature Mice Increases the Inflammatory Response to Subsequent Rhinovirus Infection: Association with Danger Signals

T. Cui, J. Hong, A. Popova, 2016, Journal of Immunology.

Differential expression of the 61 kDa and 63 kDa calmodulin-dependent phosphodiesterases in the mouse brain

C. Yan, J. K. Bentley, W. K. Sonnenburg, 1994, The Journal of neuroscience : the official journal of the Society for Neuroscience.

H. influenzae potentiates airway epithelial cell responses to rhinovirus by increasing ICAM‐1 and TLR3 expression

D. Newcomb, J. Lipuma, N. Lukacs, 2006, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Airway smooth muscle growth in asthma: proliferation, hypertrophy, and migration.

M. Hershenson, J Kelley Bentley, Marc B Hershenson, 2008, Proceedings of the American Thoracic Society.

4E-binding protein phosphorylation and eukaryotic initiation factor-4E release are required for airway smooth muscle hypertrophy.

D. Fingar, M. Abe, M. Hershenson, 2005, American journal of respiratory cell and molecular biology.

Modeling Asthma in Mice Using Rhinovirus Infection.

J. Bentley, 2022, Methods in molecular biology.

Neonatal rhinovirus induces mucous metaplasia and airways hyperresponsiveness through IL-25 and type 2 innate lymphoid cells.

J. K. Bentley, Yutein Chung, J. Lei, 2014, The Journal of allergy and clinical immunology.

Lung cells from neonates show a mesenchymal stem cell phenotype.

S. Nanua, V. Thannickal, B. Moore, 2007, American journal of respiratory and critical care medicine.

Neonatal Rhinovirus Infection Induces Mucous Metaplasia and Airways Hyperresponsiveness

J. Curtis, Ying Zhao, N. Lukacs, 2012, The Journal of Immunology.

Airway Smooth Muscle Growth in Asthma

M. Hershenson, J. Bentley, 2012 .

Transforming Growth Factor- (cid:1) Induces Airway Smooth Muscle Hypertrophy

D. Fingar, K. Bitar, M. Hershenson, 2006 .

Reduced platelet-derived growth factor receptor expression is a primary feature of human bronchopulmonary dysplasia.

Jing Lei, G. Pryhuber, T. Cui, 2014, American journal of physiology. Lung cellular and molecular physiology.

Periostin promotes fibrosis and predicts progression in patients with idiopathic pulmonary fibrosis.

F. Martinez, J. K. Bentley, K. Flaherty, 2012, American journal of physiology. Lung cellular and molecular physiology.

MDA5 and TLR3 Initiate Pro-Inflammatory Signaling Pathways Leading to Rhinovirus-Induced Airways Inflammation and Hyperresponsiveness

David J. Miller, Ying Zhao, M. Hershenson, 2011, PLoS pathogens.

The Innate Cytokines IL-25, IL-33, and TSLP Cooperate in the Induction of Type 2 Innate Lymphoid Cell Expansion and Mucous Metaplasia in Rhinovirus-Infected Immature Mice

J. K. Bentley, C. Rajput, Mingyuan Han, 2017, The Journal of Immunology.

Neonatal Periostin Knockout Mice Are Protected from Hyperoxia-Induced Alveolar Simplication

G. Pryhuber, B. Moore, A. C. Anyanwu, 2012, PloS one.

Glycogen synthase kinase-3β/β-catenin signaling regulates neonatal lung mesenchymal stromal cell myofibroblastic differentiation.

G. Pryhuber, J. Lei, M. Hershenson, 2012, American journal of physiology. Lung cellular and molecular physiology.

Autocrine production of TGF-beta1 promotes myofibroblastic differentiation of neonatal lung mesenchymal stem cells.

J. Lei, M. Hershenson, P. Bozyk, 2010, American journal of physiology. Lung cellular and molecular physiology.

Macrophage/epithelial cell CCL2 contributes to rhinovirus-induced hyperresponsiveness and inflammation in a mouse model of allergic airways disease.

T. Lewis, Yutein Chung, C. McHenry, 2013, American journal of physiology. Lung cellular and molecular physiology.

Rhinovirus Infection of Allergen-Sensitized and -Challenged Mice Induces Eotaxin Release from Functionally Polarized Macrophages

Ying Zhao, N. Lukacs, C. McHenry, 2010, The Journal of Immunology.

Rhinovirus infection induces interleukin-13 production from CD11b-positive, M2-polarized exudative macrophages.

Yutein Chung, J. Lei, M. Hershenson, 2015, American journal of respiratory cell and molecular biology.

Phosphatidylinositol 3-Kinase Is Required for Rhinovirus-induced Airway Epithelial Cell Interleukin-8 Expression*

S. Nanua, D. Newcomb, M. Hershenson, 2005, Journal of Biological Chemistry.

Enterovirus D68 infection induces IL-17-dependent neutrophilic airway inflammation and hyperresponsiveness.

C. Rajput, Mingyuan Han, J. Lei, 2018, JCI insight.

Cooperative effects of rhinovirus and TNF-α on airway epithelial cell chemokine expression

D. Newcomb, M. Hershenson, U. Sajjan, 2007 .

Rhinovirus Activates Interleukin-8 Expression via a Src/p110β Phosphatidylinositol 3-Kinase/Akt Pathway in Human Airway Epithelial Cells

D. Newcomb, M. Hershenson, U. Sajjan, 2006, Journal of Virology.

The innate cytokines IL-25, IL-33 and TSLP cooperate in the induction of ILC2 expansion and mucous metaplasia in rhinovirus-infected immature mice

C. Rajput, Mingyuan Han, Qian Wu, 2022 .

Rhinovirus colocalizes with CD68- and CD11b-positive macrophages following experimental infection in humans.

Wai-ming Lee, N. Jarjour, J. Gern, 2013, The Journal of allergy and clinical immunology.