G. Layh‐Schmitt

发表

Anion transport in oocytes of Xenopus laevis induced by expression of mouse erythroid band 3 protein‐‐encoding cRNA and of a cRNA derivative obtained by site‐directed mutagenesis at the stilbene disulfonate binding site.

H. Passow, S. Lepke, D. Bartel, 1989, The EMBO journal.

Evidence for infection with Chlamydia pneumoniae in a subgroup of patients with multiple sclerosis

A. Grau, P. Schnitzler, E. Jüttler, 2000, Annals of neurology.

Loss of bone strength in HLA-B27 transgenic rats is characterized by a high bone turnover and is mainly osteoclast-driven.

P. Zysset, L. Hofbauer, P. Pietschmann, 2015, Bone.

Two Cases of Fulminant Mycoplasma pneumoniae Pneumonia within 4 Months

J. Nollert, A. Strubel, G. Layh‐Schmitt, 2000, Infection.

HLA-B27 alters the response to tumor necrosis factor α and promotes osteoclastogenesis in bone marrow monocytes from HLA-B27-transgenic rats.

R. Colbert, G. Layh‐Schmitt, Eva Yang, 2013, Arthritis and rheumatism.

Development and Validation of a Whole-Cell Inhibition Assay for Bacterial Methionine Aminopeptidase by Surface-Enhanced Laser Desorption Ionization-Time of Flight Mass Spectrometry

A. Curnow, C. Klanke, R. Siehnel, 2005, Antimicrobial Agents and Chemotherapy.

Spatial arrangement of gene products of the P1 operon in the membrane of Mycoplasma pneumoniae

R. Herrmann, G. Layh‐Schmitt, 1994, Infection and immunity.

Spatial Arrangement ofGeneProducts oftheP1Operoninthe MembraneofMycoplasma pneumoniae

R. Herrmann, G. Layh‐Schmitt, 1994 .

Stem cell clonality and genotoxicity in hematopoietic cells: gene activation side effects should be avoidable.

C. von Kalle, B. Fehse, C. Baum, 2004, Seminars in hematology.

Visualization of the Attachment Organelle and Cytadherence Proteins of Mycoplasma pneumoniae by Immunofluorescence Microscopy

S. Seto, M. Miyata, T. Kenri, 2001, Journal of bacteriology.

Proteins complexed to the P1 adhesin of Mycoplasma pneumoniae.

M. Mann, A. Podtelejnikov, G. Layh‐Schmitt, 2000, Microbiology.

The 40- and 90-kDa membrane proteins (ORF6 gene product) of Mycoplasma pneumoniae are responsible for the tip structure formation and P1 (adhesin) association with the Triton shell.

G. Layh‐Schmitt, M. Harkenthal, 1999, FEMS microbiology letters.

Germline gain-of-function myeloid differentiation primary response gene-88 (MYD88) mutation in a child with severe arthritis.

L. Staudt, J. Tavernier, M. Gadina, 2018, The Journal of allergy and clinical immunology.

Generation and differentiation of induced pluripotent stem cells reveal ankylosing spondylitis risk gene expression in bone progenitors

C. Montagna, M. Gadina, R. Colbert, 2016, Clinical Rheumatology.

Chapter 74 – Etiology and Pathogenesis of Spondyloarthritis

R. Colbert, K. Sikora, G. Layh‐Schmitt, 2017 .

Importance of murine study design for testing toxicity of retroviral vectors in support of phase I trials.

David A. Williams, C. von Kalle, D. Witte, 2007, Molecular therapy : the journal of the American Society of Gene Therapy.

The proline-rich P65 protein of Mycoplasma pneumoniae is a component of the Triton X-100-insoluble fraction and exhibits size polymorphism in the strains M129 and FH

H. Hilbert, R. Herrmann, T. Proft, 1995, Journal of bacteriology.

A spontaneous hemadsorption-negative mutant of Mycoplasma pneumoniae exhibits a truncated adhesin-related 30-kilodalton protein and lacks the cytadherence-accessory protein HMW1

H. Hilbert, E. Pirkl, G. Layh‐Schmitt, 1995, Journal of bacteriology.

The ORF6 gene product of the P1 operon of Mycoplasma pneumoniae.

G. Layh‐Schmitt, 1993, Zentralblatt fur Bakteriologie : international journal of medical microbiology.

Localization and biochemical characterization of the ORF6 gene product of the Mycoplasma pneumoniae P1 operon

R. Herrmann, G. Layh‐Schmitt, 1992, Infection and immunity.

Immature cell populations and an erythropoiesis gene-expression signature in systemic juvenile idiopathic arthritis: implications for pathogenesis

Michael G. Barnes, B. Aronow, D. Glass, 2010, Arthritis research & therapy.

The Role of Autophagy in the Degradation of Misfolded HLA–B27 Heavy Chains

R. Colbert, Fatemeh Navid, G. Layh-Schmitt, 2018, Arthritis & rheumatology.

The interleukin-23/interleukin-17 axis in spondyloarthritis

R. Colbert, G. Layh‐Schmitt, 2008, Current opinion in rheumatology.

HLA-B27 misfolding and spondyloarthropathies.

R. Colbert, D. Sowders, M. Delay, 2009, Advances in experimental medicine and biology.

R. Himmelreich, G. Layh‐Schmitt, U. Leibfried, 1997, FEMS microbiology letters.

HLA-B27 misfolding and ankylosing spondylitis.

R. Colbert, T. Tran, G. Layh‐Schmitt, 2014, Molecular immunology.

From HLA‐B27 to spondyloarthritis: a journey through the ER

R. Colbert, M. Delay, G. Layh‐Schmitt, 2010, Immunological reviews.

HLA‑B27 misfolding and spondyloarthropathies

R. Colbert, D. Sowders, M. Delay, 2009, Prion.

Interaction of non-piliated Neisseria gonorrhoeae strain 7122 and protein IA with an epithelial cell monolayer.

T. Buchanan, S. Schmitt, G. Layh‐Schmitt, 1989, Zentralblatt fur Bakteriologie : international journal of medical microbiology.