Suelhem A. Mendoza

Transient Receptor Potential Vanilloid Type 4–Deficient Mice Exhibit Impaired Endothelium-Dependent Relaxation Induced by Acetylcholine In Vitro and In Vivo

D. Warltier, Rongshan Li, D. Gutterman, 2009, Hypertension.

H2O2-Induced Dilation in Human Coronary Arterioles: Role of Protein Kinase G Dimerization and Large-Conductance Ca2+-Activated K+ Channel Activation

Rongshan Li, D. Gebremedhin, D. Gutterman, 2012, Circulation research.

H 2 O 2 -Induced Dilation in Human Coronary Arterioles: Role of Protein Kinase G Dimerization and Large-Conductance Ca 2 (cid:1) -Activated K (cid:1) Channel Activation

Rongshan Li, D. Gebremedhin, D. Gutterman, 2012 .

H2O2 Is the Transferrable Factor Mediating Flow-Induced Dilation in Human Coronary Arterioles

D. Gutterman, David X. Zhang, Yanping Liu, 2011, Circulation research.

Activation of endothelial TRPV4 channels mediates flow-induced dilation in human coronary arterioles: role of Ca2+ entry and mitochondrial ROS signaling.

Rongshan Li, D. Gutterman, David X. Zhang, 2012, American journal of physiology. Heart and circulatory physiology.

Bradykinin-Induced Dilation of Human Coronary Arterioles Requires NADPH Oxidase–Derived Reactive Oxygen Species

K. Pritchard, D. Gutterman, B. T. Larsen, 2009, Arteriosclerosis, thrombosis, and vascular biology.

TRPV4-mediated endothelial Ca2+ influx and vasodilation in response to shear stress.

Juan Fang, Rongshan Li, D. Gutterman, 2010, American journal of physiology. Heart and circulatory physiology.

Activation of endothelial TRPV4 channels mediates flow-induced dilation in human coronary arterioles: role of Ca2+ entry and mitochondrial ROS signaling.

Rongshan Li, D. Gutterman, David X. Zhang, 2012, American journal of physiology. Heart and circulatory physiology.

Bradykinin-Induced Dilation of Human Coronary Arterioles Requires NADPH Oxidase–Derived Reactive Oxygen Species

K. Pritchard, D. Gutterman, A. Bubolz, 2009, Arteriosclerosis, thrombosis, and vascular biology.