A. M. de la Mano

N-acetylcysteine prevents intra-acinar oxygen free radical production in pancreatic duct obstruction-induced acute pancreatitis.

A. Órfão, S. Sevillano, A. M. de la Mano, 2003, Biochimica et biophysica acta.

Time-course of oxygen free radical production in acinar cells during acute pancreatitis induced by pancreatic duct obstruction.

A. Órfão, S. Sevillano, A. M. de la Mano, 2002, Biochimica et biophysica acta.

Major Pathological Mechanisms of Acute Pancreatitis Are Prevented by N-Acetylcysteine

L. Ramudo, S. Sevillano, A. M. de la Mano, 2003, Digestion.

N‐acetylcysteine induces beneficial changes in the acinar cell cycle progression in the course of acute pancreatitis

S. Sevillano, A. M. de la Mano, M. Manso, 2003, Cell proliferation.

Contribution of circulating leukocytes to cytokine production in pancreatic duct obstruction-induced acute pancreatitis in rats.

A. Órfão, L. Ramudo, S. Sevillano, 2002, Cytokine.

Heterogeneous distribution of plasma membrane glycoconjugates in pancreatic acinar cells.

S. Sevillano, A. M. de la Mano, M. Manso, 2000, Biochimica et biophysica acta.

Cholecystokinin blockade triggers earlier and enhanced intra-acinar oxygen free radical generation in acute pancreatitis induced by pancreatic duct obstruction in rats.

A. M. de la Mano, M. Manso, I. de Dios, 2003, Clinical science.

Asynchronous impairment of calcium homoeostasis in different acinar cells after pancreatic duct obstruction in rat.

A. Órfão, S. Sevillano, A. M. de la Mano, 2002, Clinical science.