Shilun Zheng

发表

Human platelets contain forms of factor V in disulfide-linkage with multimerin

F. Adam, M. Quinn-Allen, W. Kane, 2004, Thrombosis and Haemostasis.

Analyses of cellular multimerin 1 receptors: in vitro evidence of binding mediated by αΙΙbβ3 and αvβ3

F. Adam, J. Kelton, Aurelio V. Santos, 2005, Thrombosis and Haemostasis.

Platelet factor V New York: A defect in factor V distinct from that in factor V Quebec resulting in impaired prothrombinase generation

H. Weiss, B. Lages, C. Hayward, 2001, American journal of hematology.

The storage defects in grey platelet syndrome and αδ‐storage pool deficiency affect α‐granule factor V and multimerin storage without altering their proteolytic processing

Paul J. Harrison, H. Weiss, B. Lages, 2001 .

Studies of multimerin in patients with von Willebrand disease and platelet von Willebrand factor deficiency

Pille Harrison, P. Mannucci, A. Federici, 1998, British journal of haematology.

, T serotypes . rotavirus strains with distinct G ( VP 7 ) shared P ( VP 4 ) serotype between bovine Two-way cross-neutralization mediated by a

S. Ueda, G. Woode, E. Kaga, 2022 .

Analyses of cellular multimerin 1 receptors: in vitro evidence of binding mediated by alphaIIbbeta3 and alphavbeta3.

F. Adam, J. Kelton, Aurelio V. Santos, 2005, Thrombosis and haemostasis.

Platelet factor V New York: A defect in factor V distinct from that in factor V Quebec resulting in impaired prothrombinase generation

H. Weiss, B. Lages, C. Hayward, 2001, American journal of hematology.

Multimerin processing by cells with and without pathways for regulated protein secretion.

M. Pai, C. Hayward, E. Cramer, 1999, Blood.

Platelets from patients with the Quebec platelet disorder contain and secrete abnormal amounts of urokinase-type plasminogen activator.

T. Podor, G. Rivard, M. Pai, 2001, Blood.

An autosomal dominant, qualitative platelet disorder associated with multimerin deficiency, abnormalities in platelet factor V, thrombospondin, von Willebrand factor, and fibrinogen and an epinephrine aggregation defect.

J. Kelton, G. Rivard, J. Drouin, 1996, Blood.

Studies of a second family with the Quebec platelet disorder: evidence that the degradation of the alpha-granule membrane and its soluble contents are not secondary to a defect in targeting proteins to alpha-granules.

G. Rivard, C. Hayward, E. Cramer, 1997, Blood.

Studies of multimerin in human endothelial cells.

R. Stead, T. Podor, C. Hayward, 1998, Blood.