Yingzhang Lin

Three noncontiguous peptides comprise binding sites on high-molecular-weight kininogen to neutrophils.

R. Colman, A. Majluf-Cruz, S. Cooper, 1998, American journal of physiology. Heart and circulatory physiology.

Selective Inhibition of PAR4 (Protease-Activated Receptor 4)-Mediated Platelet Activation by a Synthetic Nonanticoagulant Heparin Analog

Yu Lin, S. Hung, Chin‐Chung Wu, 2019, Arteriosclerosis, thrombosis, and vascular biology.

inflammation high molecular weight kininogen in rats genetically susceptible to The mutation S511N leads to N-glycosylation and increases the cleavage of

S. Kunapuli, R. Sartor, Fengling Li, 2013 .

The mutation Ser511Asn leads to N-glycosylation and increases the cleavage of high molecular weight kininogen in rats genetically susceptible to inflammation.

R. Colman, S. Kunapuli, R. Sartor, 2003, Blood.