J. Yasufuku-Takano

发表

Mechanism of adrenomedullin-induced prolactin release from human prolactin-releasing adenoma cells.

K. Takano, T. Fujita, A. Teramoto, 2005, Endocrine journal.

Mechanism of D(2) agonist-induced inhibition of GH secretion from human GH-secreting adenoma cells.

K. Takano, T. Fujita, A. Teramoto, 2005, Endocrine journal.

Mechanisms of TRH-induced GH release (paradoxical response) in human somatotroph adenoma cells.

H. Okinaga, K. Takano, T. Fujita, 2005, Endocrine journal.

Does the prevalence of gsp mutations in GH‐secreting pituitary adenomas differ geographically or racially? Prevalence of gsp mutations in Japanese patients revisited

K. Takakura, K. Takano, T. Fujita, 2006, Clinical endocrinology.

Relationship between cytokeratin staining patterns and clinico-pathological features in somatotropinomae.

K. Sugiyama, K. Kurisu, K. Arita, 2010, European journal of endocrinology.

PRKAR1A, one of the Carney complex genes, and its locus (17q22-24) are rarely altered in pituitary tumours outside the Carney complex

J. Bertherat, W. Farrell, R. Clayton, 2002, Journal of medical genetics.

Evidence that PKA activity is constitutively activated in human GH‐secreting adenoma cells in a patient with Carney complex harbouring a PRKAR1A mutation

K. Takano, R. Osamura, T. Fujita, 2009, Clinical endocrinology.

A kindred of familial acromegaly without evidence for linkage to MEN-1 locus.

H. Shimano, N. Yahagi, N. Yamada, 2002, Endocrine journal.

Expression of pituitary tumour‐derived, N‐terminally truncated isoform of fibroblast growth factor receptor 4 (ptd‐FGFR4) correlates with tumour invasiveness but not with G‐protein alpha subunit (gsp) mutation in human GH‐secreting pituitary adenomas

T. Sano, S. Yamada, K. Takano, 2008, Clinical endocrinology.

Different G proteins mediate somatostatin‐induced inward rectifier K+ currents in murine brain and endocrine cells

T. Kozasa, Y. Nakajima, S. Nakajima, 1997, The Journal of physiology.

Morphological and physiological properties of serotonergic neurons in dissociated cultures from the postnatal rat dorsal raphe nucleus

Y. Nakajima, S. Nakajima, J. Yasufuku-Takano, 2008, Journal of Neuroscience Methods.

Gi3 mediates somatostatin-induced activation of an inwardly rectifying K+ current in human growth hormone-secreting adenoma cells.

A. Teramoto, K. Takano, T. Fujita, 1997, Endocrinology.

Gq/11 and PLC-beta 1 mediate the substance P-induced inhibition of an inward rectifier K+ channel in brain neurons.

T. Kozasa, S. Nakajima, Y. Nakajima, 1996, Journal of neurophysiology.

Heterozygous gsp mutation renders ion channels of human somatotroph adenoma cells unresponsive to growth hormone-releasing hormone.

K. Takakura, T. Fujita, S. Fukumoto, 1999, Endocrinology.

Ghrelin mRNA and GH secretagogue receptor mRNA in human GH‐producing pituitary adenomas is affected by mutations in the α subunit of G protein

K. Takano, A. Teramoto, N. Sanno, 2003, Clinical endocrinology.

Successful treatment of intractable tardive dyskinesia with botulinum toxin.

M. Sakurai, I. Kanazawa, M. Nagaoka, 1995, Journal of neurology, neurosurgery, and psychiatry.

[Pathogenesis and molecular genetics of carcinoid tumors/gastrointestinal neuroendocrine tumors].

K. Takano, J. Yasufuku-Takano, 2011, Nihon rinsho. Japanese journal of clinical medicine.

Corticotropin-releasing hormone excites adrenocorticotropin-secreting human pituitary adenoma cells by activating a nonselective cation current.

A. Teramoto, K. Takano, T. Fujita, 1996, The Journal of clinical investigation.

Effect of Ca2+ and cAMP on capacitance-measured hormone secretion in human GH-secreting adenoma cells.

N. Yamashita, K. Takano, T. Fujita, 1998, The American journal of physiology.