Nobuyo Maeda

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Homeostasis in Mice with Genetically Decreased Angiotensinogen Is Primarily by an Increased Number of Renin-producing Cells*

Hyung-Suk Kim, G. Oh, O. Smithies, 1999, The Journal of Biological Chemistry.

The Influence of Chromosomal Location on the Expression of Two Transgenes in Mice*

W. Kuziel, O. Smithies, S. Hatada, 1999, The Journal of Biological Chemistry.

Cardiovascular risk factors cause premature rarefaction of the collateral circulation and greater ischemic tissue injury

J. Faber, N. Maeda, Hua Zhang, 2015, Angiogenesis.

Inguinal Fat Compensates Whole Body Metabolic Functionality in Partially Lipodystrophic Mice with Reduced PPARγ Expression

Y. Tsai, P. Tsai, T. Chou, 2023, International journal of molecular sciences.

Altering the Expression in Mice of Genes by Modifying Their 3 (cid:1) Regions

Y. Tsai, Hyung-Suk Kim, D. Ciavatta, 2004 .

Mitochondrial DNA polymerase editing mutation, PolgD257A, reduces the diabetic phenotype of Akita male mice by suppressing appetite

T. Prolla, Hyung-Suk Kim, S. Tsutsumi, 2011, Proceedings of the National Academy of Sciences.

TIMP3 promotes the maintenance of neural stem-progenitor cells in the mouse subventricular zone

Y. Gotoh, O. Yasuda, Lingyan Fang, 2023, Frontiers in Neuroscience.

Direct evidence for the importance of endothelium-derived nitric oxide in vascular remodeling.

R. D. Rudic, W. Sessa, N. Maeda, 1998, The Journal of clinical investigation.

A mouse model for beta 0-thalassemia.

P. Detloff, B. Yang, S. Kirby, 1995, Proceedings of the National Academy of Sciences of the United States of America.

Genetic control of blood pressure and the angiotensinogen locus.

H. S. Kim, J. Hodgin, Hyung-Suk Kim, 1995, Proceedings of the National Academy of Sciences of the United States of America.

Enhanced atherosclerosis and kidney dysfunction in eNOS(-/-)Apoe(-/-) mice are ameliorated by enalapril treatment.

J. Knowles, N. Maeda, R. Reddick, 2000, The Journal of clinical investigation.

Elevated blood pressures in mice lacking endothelial nitric oxide synthase.

H. S. Kim, W. Sessa, Hyung-Suk Kim, 1996, Proceedings of the National Academy of Sciences of the United States of America.

Severe reduction in leukocyte adhesion and monocyte extravasation in mice deficient in CC chemokine receptor 2.

K. Ley, N. Maeda, W. Kuziel, 1997, Proceedings of the National Academy of Sciences of the United States of America.

Inactivation of the mouse HPRT locus by a 203-bp retroposon insertion and a 55-kb gene-targeted deletion: establishment of new HPRT-deficient mouse embryonic stem cell lines.

L. Reid, K. Edamura, T. Yoshida, 1997, Genomics.

Regulation of blood pressure by the type 1A angiotensin II receptor gene.

P. Mannon, M. Ito, T. Coffman, 1995, Proceedings of the National Academy of Sciences of the United States of America.

Single-copy transgenic mice with chosen-site integration.

N. Maeda, O. Smithies, K. Kluckman, 1996, Proceedings of the National Academy of Sciences of the United States of America.

Evidence for a novel natriuretic peptide receptor that prefers brain natriuretic peptide over atrial natriuretic peptide.

J. Knowles, M. F. Goy, N. Maeda, 2001, The Biochemical journal.

Pressure-independent enhancement of cardiac hypertrophy in natriuretic peptide receptor A-deficient mice.

J. Knowles, G. Esposito, H. Rockman, 2001, The Journal of clinical investigation.

A common human beta globin splicing mutation modeled in mice.

R. Kole, H. Sierakowska, J. Lewis, 1998, Blood.

Deletion and replacement of the mouse adult beta-globin genes by a "plug and socket" repeated targeting strategy

P. Detloff, W. Shehee, R. Langenbach, 1994, Molecular and cellular biology.

A coagulation factor IX-deficient mouse model for human hemophilia B.

N. Maeda, O. Smithies, D. Stafford, 1997, Blood.

Reduced growth, abnormal kidney structure, and type 2 (AT2) angiotensin receptor-mediated blood pressure regulation in mice lacking both AT1A and AT1B receptors for angiotensin II.

H. S. Kim, L. Audoly, M. Ito, 1998, Proceedings of the National Academy of Sciences of the United States of America.

Targeted mutation of the Hprt gene in mouse embryonic stem cells.

T. Doetschman, O. Smithies, N. Maeda, 1988, Proceedings of the National Academy of Sciences of the United States of America.

Gene targeting approaches to complex genetic diseases: atherosclerosis and essential hypertension.

N. Maeda, O. Smithies, Nobuyo Maeda, 1995, Proceedings of the National Academy of Sciences of the United States of America.

Natriuretic peptide receptor 1 expression influences blood pressures of mice in a dose-dependent manner.

M. F. Goy, N. Maeda, O. Smithies, 1998, Proceedings of the National Academy of Sciences of the United States of America.

Angiotensin-converting enzyme gene and atherosclerosis.

N. Maeda, J. Moyer, J. Krege, 1997, Arteriosclerosis, thrombosis, and vascular biology.

Hypertension, cardiac hypertrophy, and sudden death in mice lacking natriuretic peptide receptor A.

H. S. Kim, H. Rockman, R. Reddick, 1997, Proceedings of the National Academy of Sciences of the United States of America.

Isolation and expression of rat 1,25-dihydroxyvitamin D3 receptor cDNA.

H. DeLuca, N. Maeda, J. Burmester, 1988, Proceedings of the National Academy of Sciences of the United States of America.

Ventricular expression of natriuretic peptides in Npr1(-/-) mice with cardiac hypertrophy and fibrosis.

J. Knowles, V. Cameron, N. Maeda, 2002, American journal of physiology. Heart and circulatory physiology.

Npr1-regulated Gene Pathways Contributing to Cardiac Hypertrophy and Fibrosis

J. Piuhola, Nobuyo Maeda, Nicola J A Scott, 2022 .

Germ-line transmission of a planned alteration made in a hypoxanthine phosphoribosyltransferase gene by homologous recombination in embryonic stem cells.

T. Doetschman, B. Koller, O. Smithies, 1989, Proceedings of the National Academy of Sciences of the United States of America.

Limb, genital, CNS, and facial malformations result from gene/environment-induced cholesterol deficiency: further evidence for a link to sonic hedgehog.

K. Sulik, D. Dehart, L. Lanoue, 1997, American journal of medical genetics.

Differential RNA splicing and post-translational cleavages in the human salivary proline-rich protein gene system.

H. S. Kim, N. Maeda, O. Smithies, 1985, The Journal of biological chemistry.

A dominant positive and negative selectable gene for use in mammalian cells.

R. Kucherlapati, A. Skoultchi, N. Maeda, 1991, Proceedings of the National Academy of Sciences of the United States of America.

Gene transfer of endothelial nitric oxide synthase (eNOS) in eNOS-deficient mice.

N. Maeda, C. Sigmund, D. Heistad, 1999, American journal of physiology. Heart and circulatory physiology.