T. Sekiguchi

Oncostatin M suppresses generation of lymphoid progenitors in fetal liver by inhibiting the hepatic microenvironment.

A. Miyajima, H. Karasuyama, T. Kinoshita, 2001, Experimental hematology.

Cutting Edge: A Possible Role for CD4+ Thymic Macrophages as Professional Scavengers of Apoptotic Thymocytes 1

A. Miyajima, S. Koyasu, Hiroaki Ito, 2003, The Journal of Immunology.

Targeted disruption of oncostatin M receptor results in altered hematopoiesis.

M. Katsuki, Y. Hirabayashi, A. Miyajima, 2003, Blood.

Hepatic differentiation induced by oncostatin M attenuates fetal liver hematopoiesis.

Y. Ito, Yoshiaki Ito, A. Miyajima, 1999, Proceedings of the National Academy of Sciences of the United States of America.

AGM region hematopoiesis by stimulating the differentiation of endothelial cells in the Macrophage colony stimulating factor modulates the development of

A. Miyajima, T. Hara, Y. Mukouyama, 2013 .

Macrophage colony stimulating factor modulates the development of hematopoiesis by stimulating the differentiation of endothelial cells in the AGM region.

A. Miyajima, T. Hara, Y. Mukouyama, 2002, Blood.

Cultivation of aorta-gonad-mesonephros-derived hematopoietic stem cells in the fetal liver microenvironment amplifies long-term repopulating activity and enhances engraftment to the bone marrow.

A. Miyajima, T. Hara, T. Kinoshita, 2002, Blood.

Identification of podocalyxin-like protein 1 as a novel cell surface marker for hemangioblasts in the murine aorta-gonad-mesonephros region.

N. Copeland, N. Jenkins, A. Miyajima, 1999, Immunity.