Valerie A. Linck

Intracellular cholesterol stimulates ENaC by interacting with phosphatidylinositol‑4,5‑bisphosphate and mediates cyclosporine A-induced hypertension.

Zhi‐Ren Zhang, Ming‐Ming Wu, Chang Hun Song, 2019, Biochimica et biophysica acta. Molecular basis of disease.

Depletion of Cholesterol Reduces ENaC Activity by Decreasing Phosphatidylinositol-4,5-Bisphosphate in Microvilli

Zhi‐Ren Zhang, Ming‐Ming Wu, He-ping Ma, 2018, Cellular Physiology and Biochemistry.

Calcineurin A-alpha suppression drives Nuclear Factor κB-mediated NADPH oxidase-2 upregulation.

Yun Bai, R. Hoover, C. Francis, 2021, American journal of physiology. Renal physiology.

Regulation of Lung Epithelial Sodium Channels by Cytokines and Chemokines

R. Hoover, D. Eaton, B. Wynne, 2017, Front. Immunol..

Alveolar nonselective channels are ASIC1a/α-ENaC channels and contribute to AFC.

A. Alli, Megan M. Greenlee, D. Eaton, 2017, American journal of physiology. Lung cellular and molecular physiology.

Knockout of mitochondrial voltage-dependent anion channel type 3 increases reactive oxygen species (ROS) levels and alters renal sodium transport

D. Eaton, H. Bao, Q. Yue, 2017, The Journal of Biological Chemistry.

Lovastatin attenuates hypertension induced by renal tubule‐specific knockout of ATP‐binding cassette transporter A1, by inhibiting epithelial sodium channels

Zhi‐Ren Zhang, Ming‐Ming Wu, R. Sutliff, 2019, British journal of pharmacology.

RESEARCH ARTICLE Ion Channels and Transporters in Lung Function and Disease Alveolar nonselective channels are ASIC1a/ (cid:2) -ENaC channels and contribute to AFC

A. Alli, Megan M. Greenlee, D. Eaton, 2017 .