D. Rund

Two mutations in the beta-globin polyadenylylation signal reveal extended transcripts and new RNA polyadenylylation sites.

H. Kazazian, A. Oppenheim, E. Rachmilewitz, 1992, Proceedings of the National Academy of Sciences of the United States of America.

Mean corpuscular volume of heterozygotes for beta-thalassemia correlates with the severity of mutations.

A. Oppenheim, D. Filon, E. Rachmilewitz, 1992, Blood.

α‐Globin Mutations and Rearrangements in Israel: PCR‐Based Analysis Reveals Ethnic Diversity a

D. Rund, V. ORON‐KARNI, D. FILON, 1998 .

Molecular Studies of β‐Thalassemia in Israel

H. Kazazian, A. Oppenheim, D. Filon, 1990 .

Expression of beta-globin in primary erythroid progenitors of beta-thalassemia patients using an SV40-based gene delivery system.

A. Oppenheim, N. Dalyot-Herman, D. Rund, 1999, Journal of hematotherapy & stem cell research.

A novel mechanism generating short deletion/insertions following slippage is suggested by a mutation in the human alpha2-globin gene.

A. Oppenheim, D. Filon, D. Rund, 1997, Human molecular genetics.

The Relevance of Mean Corpuscular Volume of Heterozygotes to Prenatal Diagnosis of β‐Thalassemia a

A. Oppenheim, D. Filon, E. Rachmilewitz, 1990 .

Rapid detection of the common Mediterranean alpha-globin deletions/rearrangements using PCR.

A. Oppenheim, D. Filon, D. Rund, 1998, American journal of hematology.

Silent carrier β-thalassaemia due to a severe β-globin mutation interacting with other genetic elements

A. Oppenheim, D. Filon, D. Rund, 1993, European Journal of Pediatrics.

Efficient transduction of human hematopoietic cells with the human multidrug resistance gene 1 via SV40 pseudovirions.

M. Gottesman, C. Kimchi-Sarfaty, A. Oppenheim, 1998, Human gene therapy.