Dorota Gibbins

发表

Genetic dissection of Down syndrome-associated congenital heart defects using a new mouse mapping panel

V. Tybulewicz, E. Fisher, T. Mohun, 2015, eLife.

Craniofacial dysmorphology in Down syndrome is caused by increased dosage of Dyrk1a and at least three other genes

Jeremy B. A. Green, V. Tybulewicz, E. Fisher, 2022, bioRxiv.

Analysis of motor dysfunction in Down Syndrome reveals motor neuron degeneration

S. Wells, B. Kalmar, L. Greensmith, 2018, PLoS genetics.

Aging rather than aneuploidy affects monoamine neurotransmitters in brain regions of Down syndrome mouse models

M. Potier, V. Tybulewicz, E. Fisher, 2017, Neurobiology of Disease.

In vivo and ex vivo analyses of amyloid toxicity in the Tc1 mouse model of Down syndrome

V. Tybulewicz, E. Fisher, T. Maurice, 2017, Journal of psychopharmacology.

Comprehensive phenotypic analysis of the Dp1Tyb mouse strain reveals a broad range of Down syndrome-related phenotypes

S. Greenaway, A. Mallon, S. Wells, 2021, bioRxiv.

Investigating brain alterations in the Dp1Tyb mouse model of Down syndrome

B. Siow, D. Cash, A. Strydom, 2023, Neurobiology of Disease.

Congenital heart defects in Down syndrome are caused by increased dosage of DYRK1A

A. Snijders, Rifdat Aoidi, Ok-Ryul Song, 2023, bioRxiv.

Increased dosage of DYRK1A leads to congenital heart defects in a mouse model of Down syndrome

A. Snijders, M. Gutierrez, Rifdat Aoidi, 2024, Science Translational Medicine.

Dissecting the contribution of human chromosome 21 syntenic regions to recognition memory processes in adult and aged mouse models of Down syndrome

Eva Lana-Elola, Tara Canonica, Dorota Gibbins, 2024, Frontiers in behavioral neuroscience.