Alexander Koltakov

发表

α1β1 Integrin+ and Regulatory Foxp3+ T Cells Constitute Two Functionally Distinct Human CD4+ T Cell Subsets Oppositely Modulated by TNFα Blockade1

N. Amariglio, S. Ben-Horin, I. Bank, 2007, The Journal of Immunology.

Integrin and Regulatory Foxp3 T Cells Constitute Two Functionally Distinct Human CD4 T Cell Subsets Oppositely Modulated by TNF Blockade

N. Amariglio, S. Ben-Horin, V. Polevoy, 2006 .

The Effect of Blockade of Tumor Necrosis Factor α on VLA-1+T-Cells in Rheumatoid Arthritis Patients

M. Ehrenfeld, S. Ben-Horin, I. Bank, 2007, Journal of Clinical Immunology.

Lymphocytes expressing alpha1beta1 integrin (very late antigen-1) in peripheral blood of patients with arthritis are a subset of CD45RO(+) T-cells primed for rapid adhesion to collagen IV.

L. Chess, I. Bank, I. Goldstein, 2002, Clinical immunology.

Synovial VLA-1+ T cells display an oligoclonal and partly distinct repertoire in rheumatoid and psoriatic arthritis.

N. Amariglio, G. Rechavi, A. Simon, 2008, Clinical immunology.

alpha1beta1 Integrin+ and regulatory Foxp3+ T cells constitute two functionally distinct human CD4+ T cell subsets oppositely modulated by TNFalpha blockade.

N. Amariglio, S. Ben-Horin, I. Bank, 2007, Journal of Immunology.

CD4+ lymphocytes require platelets for adhesion to immobilized fibronectin in flow: Role of β1 (CD29)-, β2 (CD18)-related integrins and non-integrin receptors

N. Savion, B. Shenkman, B. Kuznik, 2006 .

Lovastatin and phospholipase Cgamma regulate constitutive and protein kinase C dependent integrin mediated interactions of human T-cells with collagen.

L. Chess, I. Bank, I. Goldstein, 2003, Cellular immunology.

Acute coronary syndromes are associated with a reduction of VLA-1+ peripheral blood T cells and their enrichment in coronary artery plaque aspirates.

H. Hod, S. Matetzky, P. Fefer, 2014, Immunobiology.

Interaction of Disease-Related Antigen-Reactive T-Cell Lines from Multiple Sclerosis Patients with Type IV Collagen: Role of Integrin VLA-1 and Effects of Irradiation

A. Achiron, G. Lavie, I. Bank, 2002, Journal of Clinical Immunology.

Platelets enhance CD4+ lymphocyte adhesion to extracellular matrix under flow conditions: Role of platelet aggregation, integrins, and non-integrin receptors

N. Savion, B. Shenkman, D. Varon, 2006, Thrombosis and Haemostasis.

CD4+ lymphocytes require platelets for adhesion to immobilized fibronectin in flow: role of beta(1) (CD29)-, beta(2) (CD18)-related integrins and non-integrin receptors.

N. Savion, B. Shenkman, B. Kuznik, 2006, Cellular Immunology.