M. Vandereyken

发表

Mechanisms of activation of innate-like intraepithelial T lymphocytes

M. Swamy, M. Vandereyken, Olivia J. James, 2020, Mucosal Immunology.

Isolation, Characterization, and Culture of Intestinal Intraepithelial Lymphocytes.

M. Swamy, M. Vandereyken, Olivia J. James, 2020, Methods in molecular biology.

Tissue environment, not ontogeny, defines intestinal intraepithelial T lymphocytes

A. Lamond, J. Hukelmann, M. Swamy, 2021, bioRxiv.

Distinct cell death pathways induced by granzymes collectively protect against intestinal Salmonella infection

J. Hinton, D. Dikovskaya, N. Wenner, 2021, bioRxiv.

Dusp3 deletion in mice promotes experimental lung tumour metastasis in a macrophage dependent manner

C. Gilles, S. Rahmouni, M. Moutschen, 2017, PloS one.

DUSP3 Phosphatase Deficiency or Inhibition Limit Platelet Activation and Arterial Thrombosis Running title: Musumeci et al.; DUSP3, a new player in arterial thrombosis

T. Mustelin, T. Chung, E. Théâtre, 2014 .

Targeting of C‐type lectin‐like receptor 2 or P2Y12 for the prevention of platelet activation by immunotherapeutic CpG oligodeoxynucleotides

S. Rahmouni, C. Lecut, C. Oury, 2017, Journal of thrombosis and haemostasis : JTH.

Dual-Specificity Phosphatase 3 Deficiency or Inhibition Limits Platelet Activation and Arterial Thrombosis

T. Mustelin, T. Chung, E. Théâtre, 2014, Circulation.

Tissue environment, not ontogeny, defines murine intestinal intraepithelial T lymphocytes

A. Lamond, D. Dikovskaya, J. Hukelmann, 2021, eLife.

Loss of adenomatous polyposis coli function renders intestinal epithelial cells resistant to the cytokine IL-22

Yu Chen, I. Näthke, I. Moraga, 2018, bioRxiv.

The Prosurvival IKK-Related Kinase IKKε Integrates LPS and IL17A Signaling Cascades to Promote Wnt-Dependent Tumor Development in the Intestine.

F. Baron, F. Greten, R. Büttner, 2016, Cancer research.

DUSP 3 Phosphatase Deficiency or Inhibition Limit Platelet Activation and Arterial Thrombosis

T. Mustelin, T. Chung, E. Théâtre, 2014 .

IL-15 and PIM kinases direct the metabolic programming of intestinal intraepithelial lymphocytes

S. Bray, J. Marchingo, F. Singh, 2021, Nature Communications.

Background—A limitation of current antiplatelet therapies is their inability to separate thrombotic events from bleeding occurrences. A better understanding of the molecular mechanisms leading to platelet activation is important for the development of improved therapies. Recently, protein tyrosine p

T. Mustelin, T. Chung, E. Théâtre, 2015 .

Dual-Specificity Phosphatase 3 Deletion Protects Female, but Not Male, Mice from Endotoxemia-Induced and Polymicrobial-Induced Septic Shock

C. Libert, S. Rahmouni, M. Moutschen, 2017, The Journal of Immunology.

DUSP3 Genetic Deletion Confers M2-like Macrophage–Dependent Tolerance to Septic Shock

T. Mustelin, E. Théâtre, C. Libert, 2015, The Journal of Immunology.

DUSP3/VHR is a pro-angiogenic atypical dual-specificity phosphatase

Maud Martin, F. Dequiedt, T. Mustelin, 2014, Molecular Cancer.

Mechanisms of activation of innate-like intraepithelial T lymphocytes

M. Swamy, M. Vandereyken, Olivia J. James, 2020, Mucosal Immunology.