R. Viana

发表

TUDCA, a Bile Acid, Attenuates Amyloid Precursor Protein Processing and Amyloid-β Deposition in APP/PS1 Mice

R. D'Hooge, C. Rodrigues, C. Rodrigues, 2012, Molecular Neurobiology.

Naphtho[2,3-d]isoxazole-4,9-dione-3-carboxylates: potent, non-cytotoxic, antiapoptotic agents.

Maria M. M. Santos, C. Rodrigues, R. Moreira, 2009, Chemico-biological interactions.

Modulation of amyloid-β peptide-induced toxicity through inhibition of JNK nuclear localization and caspase-2 activation.

C. Steer, C. Rodrigues, R. Ramalho, 2010, Journal of Alzheimer's disease : JAD.

Bile acids: regulation of apoptosis by ursodeoxycholic acid

C. Steer, C. Rodrigues, J. Amaral, 2009, Journal of Lipid Research.

Amyloid-β peptide-induced secretion of endoplasmic reticulum chaperone glycoprotein GRP94.

C. Steer, C. Rodrigues, R. Viana, 2011, Journal of Alzheimer's disease : JAD.

Apoptosis signaling associated with amyloid β-induced cellular dysfunction and degeneration : relevance for Alzheimer’s disease

R. Viana, Ricardo J. S. Viana, 2010 .

Bile acids and apoptosis modulation: an emerging role in experimental Alzheimer's disease.

C. Steer, W. Low, C. Rodrigues, 2008, Trends in molecular medicine.

Endoplasmic Reticulum Enrollment in Alzheimer’s Disease

C. Rodrigues, C. Rodrigues, A. Nunes, 2012, Molecular Neurobiology.

TUDCA, a Bile Acid, Attenuates Amyloid Precursor Protein Processing and Amyloid-β Deposition in APP/PS1 Mice

R. D'Hooge, C. Rodrigues, J. Amaral, 2012, Molecular Neurobiology.

Contrasting apoptotic responses of conjugated linoleic acid in the liver of obese Zucker rats fed palm oil or ovine fat.

C. Rodrigues, J. Prates, R. Bessa, 2011, Prostaglandins, leukotrienes, and essential fatty acids.

Endoplasmic Reticulum Enrollment in Alzheimer’s Disease

C. Rodrigues, A. Nunes, R. Viana, 2012, Molecular Neurobiology.

Tauroursodeoxycholic acid prevents E22Q Alzheimer’s Aβ toxicity in human cerebral endothelial cells

J. Meyerson, J. Ghiso, C. Rodrigues, 2009, Cellular and Molecular Life Sciences.

Apoptosis in Transgenic Mice Expressing the P301L Mutated Form of Human Tau

C. Steer, W. Low, C. Rodrigues, 2008, Molecular medicine.