Shenglong Chen

The nonpeptide AVE0991 attenuates myocardial hypertrophy as induced by angiotensin II through downregulation of transforming growth factor-β1/Smad2 expression

Hong Ma, Jian-gui He, Yu-gang Dong, 2010, Heart and Vessels.

Acute myocardial ischemia directly modulates the expression of brain natriuretic peptide at the transcriptional and translational levels via inflammatory cytokines.

Jian-gui He, Yi-li Chen, Shenglong Chen, 2011, European journal of pharmacology.

B‐type natriuretic peptide attenuates cardiac hypertrophy via the transforming growth factor‐β1/smad7 pathway in vivo and in vitro

Fengjuan Yao, Jian-gui He, Yu-gang Dong, 2010, Clinical and Experimental Pharmacology and Physiology.

Endogenous BNP attenuates cardiomyocyte hypertrophy induced by Ang II via p38 MAPK/Smad signaling.

Lingling Wu, Yugang Dong, Yili Chen, 2014, Die Pharmazie.

AVE0991, a Nonpeptide Compound, Attenuates Angiotensin II-Induced Vascular Smooth Muscle Cell Proliferation via Induction of Heme Oxygenase-1 and Downregulation of p-38 MAPK Phosphorylation

Chen Sheng-Long, Wu Yan-Xin, Huang Yi-yi, 2012, International journal of hypertension.

Effect of long-term B-type natriuretic peptide treatment on left ventricular remodeling and function after myocardial infarction in rats.

Zhongkai Wu, Lichun Wang, Yili Chen, 2009, European journal of pharmacology.

B‐type natriuretic peptide attenuates cardiac hypertrophy via the transforming growth factor‐β1/smad7 pathway in vivo and in vitro

Fengjuan Yao, Jian-gui He, Yu-gang Dong, 2010, Clinical and experimental pharmacology & physiology.