Monica T Yu Kimata

ICAM-3 influences human immunodeficiency virus type 1 replication in CD4(+) T cells independent of DC-SIGN-mediated transmission.

R. Arora, J. Kimata, J. Biggins, 2007, Virology.

Highly Pathogenic Simian Immunodeficiency Virus mne Variants That Emerge during the Course of Infection Evolve Enhanced Infectivity and the Ability To Downregulate CD4 but Not Class I Major Histocompatibility Complex Antigens

J. Kimata, J. Biggins, Monica T Yu Kimata, 2002, Journal of Virology.

Vif Substitution Enables Persistent Infection of Pig-Tailed Macaques by Human Immunodeficiency Virus Type 1

R. Arora, Shiu-Lok Hu, P. Zhou, 2011, Journal of Virology.

Changes in simian immunodeficiency virus reverse transcriptase alleles that appear during infection of macaques enhance infectivity and replication in CD4+ T cells.

J. Kimata, Tasha Biesinger, Monica T Yu Kimata, 2008, Virology.

Relative replication capacity of phenotypic SIV variants during primary infections differs with route of inoculation

J. Allan, J. Kimata, B. Wilson, 2010, Retrovirology.

Capture and Transfer of Simian Immunodeficiency Virus by Macaque Dendritic Cells Is Enhanced by DC-SIGN

M. Colonna, M. Cella, J. Allan, 2002, Journal of Virology.

Domains of macaque DC-SIGN essential for capture and transfer of simian immunodeficiency virus.

J. Kimata, J. Biggins, Monica T Yu Kimata, 2004, Virology.