D. Sacks

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Proteinmethyltransferase 7 deficiency in Leishmania major increases neutrophil associated pathology inmurinemodel

P. Volf, A. Cruz, Lucas, 2021 .

Tracking antigen‐specific CD4+ T cells throughout the course of chronic Leishmania major infection in resistant mice

M. Jenkins, C. Karp, M. Pepper, 2013, European journal of immunology.

The Nlrp3 inflammasome, IL‐1β, and neutrophil recruitment are required for susceptibility to a nonhealing strain of Leishmania major in C57BL/6 mice

F. Tacchini-Cottier, A. Romano, K. Mayer-Barber, 2016, European journal of immunology.

The impact of vector‐mediated neutrophil recruitment on cutaneous leishmaniasis

N. Peters, D. Sacks, 2009, Cellular microbiology.

Resistance Against Leishmania major Infection Depends on Microbiota-Guided Macrophage Activation

L. Vieira, M. E. Lopes, M. Carneiro, 2021, Frontiers in Immunology.

Targeted gene deletion of Leishmania major genes encoding developmental stage‐specific leishmanolysin (GP63)

W. McMaster, G. Modi, P. Joshi, 1998, Molecular microbiology.

Experimental Hybridization in Leishmania: Tools for the Study of Genetic Exchange

T. R. Ferreira, D. Sacks, T. Ferreira, 2022, Pathogens.

Primate Model of Cutaneous Leishmaniasis Human IL-12 and Alum as Adjuvants in a Protective Immunity Using Recombinant

J. Sypek, A. Gam, R. Kenney, 1999 .

CD4+CD25+ regulatory T cells control Leishmania major persistence and immunity

Y. Belkaid, E. Shevach, C. Piccirillo, 2002, Nature.

Developmental modification of lipophosphoglycan during the differentiation of Leishmania major promastigotes to an infectious stage.

M. Ferguson, M. McConville, S. Turco, 1992, The EMBO journal.

Reassessment of Immune Correlates in Human Visceral Leishmaniasis as Defined by Cytokine Release in Whole Blood

R. Kumar, O. Singh, S. Sundar, 2012, Clinical and Vaccine Immunology.

This information is current as Leishmania infantum Visceral Infection with Mediates Heterologous Protection against Leishmania major Cutaneous Infection with

N. Peters, A. Romano, Nicole A. Doria, 2015 .

Analysis of cytokine production by inflammatory mouse macrophages at the single‐cell level: selective impairment of IL‐12 induction in Leishmania ‐infected cells

Yasmine Belkaid, Y. Belkaid, B. Butcher, 1998, European journal of immunology.

Characterization of a blood activated chitinolytic system in the midgut of the sand fly vectors Lutzomyia longipalpis and Phlebotomus papatasi

S. Kamhawi, J. Valenzuela, M. Joshi, 2005, Insect molecular biology.

Leishmania–sand fly interactions controlling species‐specific vector competence

D. Sacks, D. Sacks, 2001, Cellular microbiology.

Evolution of Lesion Formation, Parasitic Load, Immune Response, and Reservoir Potential in C57BL/6 Mice following High- and Low-Dose Challenge with Leishmania major

G. Modi, M. Doherty, Govind Modi, 2000, Infection and Immunity.

Trypanosome‐induced suppression of anti‐parasite responses during experimental African trypanosomiasis

B. Askonas, D. Sacks, Brigitte A. Askonas, 1980, European journal of immunology.

NOX2-Derived Reactive Oxygen Species Control Inflammation during Leishmania amazonensis Infection by Mediating Infection-Induced Neutrophil Apoptosis

A. Debrabant, N. Peters, L. Vieira, 2018, The Journal of Immunology.

Deficiency in β1,3-Galactosyltransferase of a Leishmania major Lipophosphoglycan Mutant Adversely Influences the Leishmania-Sand Fly Interaction*

B. Butcher, S. Turco, P. Pimenta, 1996, The Journal of Biological Chemistry.

Dermis resident macrophages orchestrate localized ILC2-eosinophil circuitries to maintain their M2-like properties and promote non-healing cutaneous leishmaniasis

J. Kabat, O. Kamenyeva, B. Kelsall, 2023, Research square.

Animal Models for the Analysis of Immune Responses to Leishmaniasis

P. Melby, D. Sacks, D. Sacks, 2001, Current protocols in immunology.

Stress conditions promote Leishmania hybridization in vitro marked by expression of the ancestral gamete fusogen HAP2 as revealed by single-cell RNA-seq

A. Paun, Isabelle Louradour, D. Sacks, 2022, eLife.

Self-Hybridization in Leishmania major

D. Dobson, S. Beverley, E. Inbar, 2022, mBio.

Stress conditions promote the mating competency of Leishmania promastigotes in vitro marked by expression of the ancestral gamete fusogen HAP2

N. Karunaweera, A. Paun, Isabelle Louradour, 2021 .

Animal models for the analysis of immune responses to leishmaniasis.

P. Melby, D. Sacks, D. Sacks, 2015, Current protocols in immunology.

Human visceral leishmaniasis is not associated with expansion or accumulation of Foxp3+ CD4 cells in blood or spleen.

Rajiv Kumar, S. Sundar, V. Prajapati, 2010, Parasite immunology.

CR1, the C3b receptor, mediates binding of infective Leishmania major metacyclic promastigotes to human macrophages.

B. F. Hall, K. Joiner, D. Sacks, 1989, Journal of immunology.

Molecular aspects of parasite-vector and vector-host interactions in leishmaniasis.

S. Kamhawi, D. Sacks, D Sacks, 2001, Annual review of microbiology.

The role of the lipophosphoglycan of Leishmania in vector competence

D. Sacks, S. Turco, E. Rowton, 1994, Parasitology.

IL-4- and IL-4 receptor-deficient BALB/c mice reveal differences in susceptibility to Leishmania major parasite substrains.

W. Paul, D. Sacks, N. Noben-Trauth, 1999, Journal of immunology.

Membrane fractions of trypanosomes mimic the immunosuppressive and mitogenic effects of living parasites on the host

C. Clayton, D. Sacks, B. Askonas, 1979, Parasite immunology.

Metacyclogenesis is a major determinant of Leishmania promastigote virulence and attenuation

D. Sacks, R. D. da Silva, Rosângela, 1987, Infection and immunity.

Genes selectively expressed in the infectious (metacyclic) stage of Leishmania major promastigotes encode a potential basic-zipper structural motif.

D. Sacks, S. Heath, D. Sacks, 1992, Molecular and biochemical parasitology.

Targeted gene deletion in Leishmania major identifies leishmanolysin (GP63) as a virulence factor.

W. McMaster, B. Kelly, S. Kamhawi, 2002, Molecular and biochemical parasitology.

The impact of vector‐mediated neutrophil recruitment on cutaneous leishmaniasis

N. Peters, D. Sacks, 2009, Cellular microbiology.

The Nlrp3 inflammasome, IL‐1β, and neutrophil recruitment are required for susceptibility to a nonhealing strain of Leishmania major in C57BL/6 mice

F. Tacchini-Cottier, A. Romano, K. Mayer-Barber, 2016, European Journal of Immunology.

The role of antigen and IL-12 in sustaining Th1 memory cells in vivo: IL-12 is required to maintain memory/effector Th1 cells sufficient to mediate protection to an infectious parasite challenge.

N. Glaichenhaus, S. Gurunathan, R. Seder, 2000, Proceedings of the National Academy of Sciences of the United States of America.

Protective immunity using recombinant human IL-12 and alum as adjuvants in a primate model of cutaneous leishmaniasis.

D. Sacks, J. Sypek, A. Gam, 1999, Journal of immunology.

Protection against cutaneous leishmaniasis resulting from bites of uninfected sand flies.

Y. Belkaid, S. Kamhawi, G. Modi, 2000, Science.

Identification of antigens recognized by T cells in human leishmaniasis: analysis of T-cell clones by immunoblotting

P. Melby, D. Sacks, D. Sacks, 1989, Infection and immunity.

Leishmania tropica: the identification and purification of metacyclic promastigotes and use in establishing mouse and hamster models of cutaneous and visceral disease.

C. Jaffe, D. Sacks, F. Neva, 1998, Experimental parasitology.