T. Izumi

发表

Anti-β2-glycoprotein I Antibody-mediated Inhibition of Activated Protein C Requires Binding of β2-glycoprotein I to Phospholipids

G. M. Iverson, T. Ortel, Z. Su, 2002, Thrombosis and Haemostasis.

Novel Deletion and Insertion Mutations Cause Splicing Defects, Leading to Severe Reduction in mRNA Levels of the A Subunit in Severe Factor XIII Deficiency

T. Saito, J. Takamatsu, H. Saito, 1998, Thrombosis and Haemostasis.

Molecular and genetic mechanisms of factor XIII A subunit deficiency.

A. Ichinose, M. Souri, T. Izumi, 2000, Seminars in thrombosis and hemostasis.

Arg260‐Cys mutation in severe factor XIII deficiency: conformational change of the A subunit is predicted by molecular modelling and mechanics

J. Takamatsu, H. Umeyama, H. Saito, 1998, British journal of haematology.

Type I factor XIII deficiency is caused by a genetic defect of its b subunit: insertion of triplet AAC in exon III leads to premature termination in the second Sushi domain.

A. Tosetto, A. Girolami, F. Rodeghiero, 1996, Blood.

The Normal and Abnormal Genes of the a and b Subunits in Coagulation Factor XIII

A. Ichinose, T. Izumi, T. Hashiguchi, 1996, Seminars in thrombosis and hemostasis.

Molecular Pathology of Various Types of Factor XIII Deficiencies

A. Ichinose, T. Izumi, T. Hashiguchi, 1996 .

Glycosylation engineering of therapeutic IgG antibodies: challenges for the safety, functionality and efficacy

P. Rudd, Kenji Yamamoto, Toshihiko Katoh, 2017, Protein & Cell.

Antiphospholipid antibodies after surgical exposure to topical bovine thrombin.

T. Ortel, J. Lawson, E. Thames, 2002, The Journal of laboratory and clinical medicine.

Fine Mapping of Inhibitory Anti-factor V Antibodies Using Factor V C2 Domain Mutants

T. Ortel, W. Bode, S. Macedo-Ribeiro, 2001, Thrombosis and Haemostasis.

Prothrombin and β2-glycoprotein I frequently contribute to antiphospholipid antibody interactions with phospholipids and the generation of abnormal waveform profiles in coagulation assays

T. Ortel, Z. Su, T. Izumi, 2003, Thrombosis and Haemostasis.

A Founder Effect Is Proposed for Factor XIII B Subunit Deficiency Caused by the Insertion of Triplet AAC in Exon III Encoding the Second Sushi Domain

A. Girolami, A. Ichinose, M. Souri, 1998, Thrombosis and Haemostasis.

Ala601‐Thr type dysplasminogenaemia genetically diagnosed in patients with retinochoroidal vascular disorders

A. Ichinose, M. Murata, M. Nakagawa, 1997, British journal of haematology.