Ilham El Khattabi

发表

Compensatory Response by Late Embryonic Tubular Epithelium to the Reduction in Pancreatic Progenitors

S. Bonner-Weir, Arun J Sharma, K. Yasuda, 2015, PloS one.

Differentiation of pancreatic endocrine progenitors reversibly blocked by premature induction of MafA.

S. Bonner-Weir, Arun J Sharma, K. He, 2014, Developmental biology.

p38 MAPK is a major regulator of MafA protein stability under oxidative stress.

S. Bonner-Weir, Arun J Sharma, D. R. Laybutt, 2009, Molecular endocrinology.

The regulation of IGFs and IGFBPs by prolactin in primary culture of fetal rat hepatocytes is influenced by maternal malnutrition.

B. Reusens, C. Remacle, Ilham El Khattabi, 2006, American journal of physiology. Endocrinology and metabolism.

A switch from MafB to MafA expression accompanies differentiation to pancreatic beta-cells.

S. Bonner-Weir, Arun J Sharma, T. Salameh, 2006, Developmental biology.

Proper activation of MafA is required for optimal differentiation and maturation of pancreatic β-cells.

Arun J Sharma, Ilham El Khattabi, 2015, Best practice & research. Clinical endocrinology & metabolism.

Mutations at the BLK locus linked to maturity onset diabetes of the young and β-cell dysfunction

S. Bonner-Weir, Arun J Sharma, S. Rich, 2009, Proceedings of the National Academy of Sciences.

Thyroid Hormone Promotes Postnatal Rat Pancreatic β-Cell Development and Glucose-Responsive Insulin Secretion Through MAFA

P. Larsen, S. Bonner-Weir, Arun J Sharma, 2013, Diabetes.

Trimeprazine increases IRS2 in human islets and promotes pancreatic β cell growth and function in mice.

S. Bonner-Weir, Arun J Sharma, M. White, 2016, JCI insight.

β-cell preservation and regeneration for diabetes treatment: where are we now?

Arun J Sharma, A. Kapoor, Ilham El Khattabi, 2012, Diabetes management.

Preventing p38 MAPK-mediated MafA degradation ameliorates β-cell dysfunction under oxidative stress.

Arun J Sharma, Ilham El Khattabi, 2013, Molecular endocrinology.