Kyungdeok Kim

发表

Tanc2-dependent direct and regulated mTOR inhibition balances mTORC1/2 signaling in developing mouse and human neurons

C. Yook, W. Heo, Eunjoon Kim, 2020 .

NGL-2 Deletion Leads to Autistic-like Behaviors Responsive to NMDAR Modulation.

Y. Bae, Eunjoon Kim, S. Um, 2018, Cell reports.

Early postnatal serotonin modulation prevents adult-stage deficits in Arid1b-deficient mice through synaptic transcriptional reprogramming

Eunjoon Kim, Hyojin Kang, Sunjoo Ahn, 2022, Nature Communications.

Author Correction: Tanc2-mediated mTOR inhibition balances mTORC1/2 signaling in the developing mouse brain and human neurons

C. Yook, W. Heo, Eunjoon Kim, 2021, Nature Communications.

Social deficit is a major feature of neuropsychiatric disorders, including autism

Se-Bum Paik, Eunjoon Kim, Woohyun Kim, 2022 .

Parenchymal border macrophages regulate the flow dynamics of the cerebrospinal fluid

Nick C Fox, Alyssa R. Gonzalez, A. Fagan, 2022, Nature.

Trans-synaptic zinc mobilization improves social interaction in two mouse models of autism through NMDAR activation

Eunjoon Kim, J. Koh, Changuk Chung, 2015, Nature Communications.

Early correction of synaptic long-term depression improves abnormal anxiety-like behavior in adult GluN2B-C456Y-mutant mice

Y. Bae, P. Paoletti, Eunjoon Kim, 2020, PLoS biology.

NGL-3 in the regulation of brain development, Akt/GSK3b signaling, long-term depression, and locomotive and cognitive behaviors

Eunjoon Kim, Hyun Kim, Esther Yang, 2019, PLoS biology.

A TBR1-K228E Mutation Induces Tbr1 Upregulation, Altered Cortical Distribution of Interneurons, Increased Inhibitory Synaptic Transmission, and Autistic-Like Behavioral Deficits in Mice

C. Yook, Eunjoon Kim, Hyojin Kang, 2019, Front. Mol. Neurosci..

Lrfn2-Mutant Mice Display Suppressed Synaptic Plasticity and Inhibitory Synapse Development and Abnormal Social Communication and Startle Response

Y. Bae, Eunjoon Kim, Doyoun Kim, 2018, The Journal of Neuroscience.

Low-Level Brain Somatic Mutations Are Implicated in Schizophrenia

M. Webster, Sanghyeon Kim, Jun Sung Park, 2021, Biological Psychiatry.

Tanc2-mediated mTOR inhibition balances mTORC1/2 signaling in the developing mouse brain and human neurons

C. Yook, W. Heo, Eunjoon Kim, 2021, Nature Communications.

Scn2a Haploinsufficiency in Mice Suppresses Hippocampal Neuronal Excitability, Excitatory Synaptic Drive, and Long-Term Potentiation, and Spatial Learning and Memory

Eunjoon Kim, Doyoun Kim, Hyun Kim, 2019, Front. Mol. Neurosci..

Hyperactive ACC-MDT Pathway Suppresses Prepulse Inhibition in Mice.

Eunjoon Kim, Yangsik Kim, Kyungdeok Kim, 2020, Schizophrenia bulletin.

IRSp53 Deletion in Glutamatergic and GABAergic Neurons and in Male and Female Mice Leads to Distinct Electrophysiological and Behavioral Phenotypes

Eunjoon Kim, Hyun Kim, Yangsik Kim, 2020, Frontiers in Cellular Neuroscience.

Presynaptic PTPσ regulates postsynaptic NMDA receptor function through direct adhesion-independent mechanisms

Y. Bae, Eunjoon Kim, Doyoun Kim, 2020, eLife.

GABAergic neuronal IL-4R mediates T cell effect on memory

Jasmin Herz, I. Smirnov, J. Kipnis, 2021, Neuron.

Synaptic scaffolding molecule binds to and regulates vasoactive intestinal polypeptide type-1 receptor in epithelial cells.

Min Goo Lee, Hoguen Kim, Eunjoon Kim, 2009, Gastroenterology.

Corrigendum: IRSp53 Deletion in Glutamatergic and GABAergic Neurons and in Male and Female Mice Leads to Distinct Electrophysiological and Behavioral Phenotypes

Eunjoon Kim, Hyun Kim, Yangsik Kim, 2021, Frontiers in Cellular Neuroscience.

SALM4 negatively regulates NMDA receptor function and fear memory consolidation

Eunjoon Kim, S. Um, Hyun Kim, 2021, Communications Biology.

Stress induces behavioral abnormalities by increasing expression of phagocytic receptor, MERTK, in astrocytes to promote synapse phagocytosis.

Eunjoon Kim, Won-Suk Chung, Youkyeong Gloria Byun, 2023, Immunity.

Loss of Katnal2 leads to ependymal ciliary hyperfunction and autism-related phenotypes in mice

Esther Yang, Suho Lee, Miram Shin, 2024, PLoS biology.