M. Blom

Granulocyte-macrophage colony-stimulating factor, interleukin-3 (IL-3), and IL-5 greatly enhance the interaction of human eosinophils with opsonized particles by changing the affinity of complement receptor type 3.

L. Koenderman, D. Roos, A. Verhoeven, 1994, Blood.

Eosinophils isolated with two different methods show different characteristics of activation.

E. Knol, D. Roos, A. Verhoeven, 1995, Journal of immunological methods.

Transient exposure of human eosinophils to the protein kinase C inhibitors CGP39‐360, CGP41‐251, and CGP44‐800 leads to priming of the respiratory burst induced by opsonized particles

J. Lammers, L. Koenderman, A. Verhoeven, 1993, Journal of leukocyte biology.

Characteristics of CR3-mediated aggregation in human eosinophils: effect of priming by platelet-activating factor.

T. Kuijpers, L. Koenderman, D. Roos, 1991, The Journal of allergy and clinical immunology.

Priming of human eosinophils by platelet-activating factor enhances the number of cells able to bind and respond to opsonized particles.

D. Roos, A. Verhoeven, A. Tool, 1992, Journal of immunology.

Human eosinophils express, relative to other circulating leukocytes, large amounts of secretory 14-kD phospholipase A2.

C. Hack, J. Calafat, E. Knol, 1998, Blood.

Release of platelet-activating factor is important for the respiratory burst induced in human eosinophils by opsonized particles.

L. Koenderman, D. Roos, A. Verhoeven, 1992, Blood.

Eosinophils and Platelet-Activating Factor

L. Koenderman, A. Tool, A. Verhoeven, 2019, Eosinophils in Allergy and Inflammation.