J. Querol

The BRD4S-LOXL2-MED1 interaction at the forefront of cell cycle transcriptional control in triple-negative breast cancer

Lorena Espinar, J. Arribas, L. Serrano, 2022, bioRxiv.

LOXL2-mediated H3K4 oxidation reduces chromatin accessibility in triple-negative breast cancer cells

A. Gonzalez-Perez, P. Nuciforo, J. Yélamos, 2019, Oncogene.

Reduction of LOXL2-mediated H3K4 oxidation increases chromatin accessibility and promotes chemosensitivity of triple-negative breast cancer cells

A. Gonzalez-Perez, P. Nuciforo, J. Yélamos, 2018, bioRxiv.

GEMC1 and MCIDAS interactions with SWI/SNF complexes regulate the multiciliated cell-specific transcriptional program

Camille Stephan-Otto Attolini, X. Salvatella, B. Raught, 2020, bioRxiv.

Maintaining oxidized H3 in heterochromatin is required for the oncogenic capacity of triple-negative breast cancer cells

E. Blanco, P. Nuciforo, R. Dienstmann, 2020, bioRxiv.

Fascin-1 expression is associated with neuroendocrine prostate cancer and directly suppressed by androgen receptor

A. Carnot, M. Duterque-Coquillaud, Carine Delliaux, 2023, British Journal of Cancer.

LOXL2‐mediated chromatin compaction is required to maintain the oncogenic properties of triple‐negative breast cancer cells

R. Dienstmann, V. Peg, L. Di Croce, 2024, The FEBS journal.

Interactions between BRD4S, LOXL2, and MED1 drive cell cycle transcription in triple‐negative breast cancer

S. Sdelci, Anestis Gkanogiannis, Chiara Cannatá, 2023, EMBO molecular medicine.