E. Pedrosa

发表

Genetic and experimental evidence for the involvement of the CD6 lymphocyte receptor in psoriasis

J. Romaní, J. Carrascosa, C. Ferrándiz, 2018, Cellular & Molecular Immunology.

Genetically defined variants of toll-like receptors 3, 7 and 9 as phenotype and risk modifier factors for psoriasis.

J. Romaní, J. Carrascosa, C. Ferrándiz, 2017, Journal of dermatological science.

Copy number variation in the CCL4L gene is associated with susceptibility to acute rejection in lung transplantation

M. Juan, R. Faner, R. Colobran, 2009, Genes and Immunity.

Copy number variation in chemokine superfamily: the complex scene of CCL3L–CCL4L genes in health and disease

M. Juan, R. Colobran, L. Carretero-Iglesia, 2010, Clinical and experimental immunology.

CCL4L polymorphisms and CCL4/CCL4L serum levels are associated with psoriasis severity.

M. Juan, R. Faner, R. Colobran, 2011, The Journal of investigative dermatology.

Population structure in copy number variation and SNPs in the CCL4L chemokine gene

D. Comas, M. Juan, J. Bertranpetit, 2008, Genes and Immunity.

Genetic and experimental evidence for the involvement of the CD6 lymphocyte receptor in psoriasis

J. Romaní, J. Carrascosa, C. Ferrándiz, 2018, Cellular & Molecular Immunology.

The role of Fcγ receptor polymorphisms in the response to anti–tumor necrosis factor therapy in psoriasis A pharmacogenetic study.

J. Carrascosa, C. Ferrándiz, M. Alsina-Gibert, 2013, JAMA dermatology.

Disruption of striatal glutamatergic transmission induced by mutant huntingtin involves remodeling of both postsynaptic density and NMDA receptor signaling

A. Giralt, J. Alberch, J. Canals, 2008, Neurobiology of Disease.

Population structure in copy number variation and SNPs in the CCL4L chemokine gene

D. Comas, M. Juan, J. Bertranpetit, 2008, Genes and Immunity.