L. Jones

Chronic cocaine treatment enhances the responsiveness of the left anterior descending coronary artery and the femoral artery to vasoactive substances.

R. Tackett, L. Jones, 1990, The Journal of pharmacology and experimental therapeutics.

Characterization of coronary vasoconstriction produced by rostral ventrolateral medulla stimulation in rats.

M. Brody, L. Jones, 1992, The American journal of physiology.

Interaction of propranolol with central serotonergic neurons.

R. Tackett, L. Jones, 1988, Life sciences.

Sympathetic nervous system mediates central corticotropin-releasing factor induced suppression of natural killer cytotoxicity.

M. Irwin, R. Hauger, M. Provencio, 1990, The Journal of pharmacology and experimental therapeutics.

Differential routes of cocaine administration indicate a peripheral cardiotoxic action

R. Tackett, L. Jones, 1991, Pharmacology, Biochemistry and Behavior.

Enhanced pressor response to cocaine in SHR is mediated through peripheral alpha receptors

R. Tackett, L. Jones, 1990 .

Central corticotropin releasing factor reduces natural cytotoxicity. Time course of action.

M. Irwin, R. Hauger, K. Britton, 1989, Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology.

Catecholaminergic and opioidergic mechanisms involved in the hypotensive response of pindolol.

R. Tackett, L. Jones, 1989, European journal of pharmacology.

Central mechanisms of action involved in cocaine-induced tachycardia.

R. Tackett, L. Jones, 1990, Life sciences.

Coronary blood flow in rats is dependent on the release of vascular nitric oxide.

M. Brody, L. Jones, 1992, The Journal of pharmacology and experimental therapeutics.