Amy L. Myers

发表

The Pancreatic Tumor Microenvironment Buffers Redox Imbalance Imposed by Disrupted Mitochondrial Metabolism

Z. Nwosu, Lin Lin, H. Wen, 2020 .

The Pancreatic Tumor Microenvironment Compensates for Loss of GOT2

Z. Nwosu, D. Piwnica-Worms, C. Lyssiotis, 2020, bioRxiv.

Metabolic requirement for GOT2 in pancreatic cancer depends on environmental context

Z. Nwosu, D. Piwnica-Worms, H. Wen, 2022, eLife.

Inhibition of angiogenic initiation and disruption of newly established human vascular networks by juice from Morinda citrifolia (noni)

C. Hornick, E. Woltering, H. Sadowska-Krowicka, 2004, Angiogenesis.

Osteopontin (OPN/SPP1) isoforms collectively enhance tumor cell invasion and dissemination in esophageal adenocarcinoma

Thomas D. Wang, D. Beer, Lin Lin, 2015, Oncotarget.

GOT1 inhibition promotes pancreatic cancer cell death by ferroptosis

Nupur K. Das, Stephen A. Sastra, Carmine F. Palermo, 2021, Nature Communications.

GOT1 Inhibition Primes Pancreatic Cancer for Ferroptosis through the Autophagic Release of Labile Iron

Stephen A. Sastra, Carmine F. Palermo, Z. Nwosu, 2020, bioRxiv.

IGFBP2 modulates the chemoresistant phenotype in esophageal adenocarcinoma

D. Beer, Lin Lin, Dafydd G. Thomas, 2015, Oncotarget.

Tissue of origin dictates GOT1 dependence and confers synthetic lethality to radiotherapy

Z. Nwosu, Lin Lin, Ho-Joon Lee, 2019, Cancer & Metabolism.

Therapeutic targeting of differentiation state-dependent metabolic vulnerabilities in DIPG

Z. Nwosu, S. Venneti, C. Lyssiotis, 2022, bioRxiv.