Craig W. Younce

Exendin-4 improves cardiac function in mice overexpressing monocyte chemoattractant protein-1 in cardiomyocytes.

J. Ayala, P. Kolattukudy, Layton H Smith, 2014, Journal of molecular and cellular cardiology.

MCP-1 Induced Protein Promotes Adipogenesis via Oxidative Stress, Endoplasmic Reticulum Stress and Autophagy

P. Kolattukudy, Craig W. Younce, 2012, Cellular Physiology and Biochemistry.

Exendin-4 attenuates high glucose-induced cardiomyocyte apoptosis via inhibition of endoplasmic reticulum stress and activation of SERCA2a.

J. Ayala, M. Burmeister, Craig W Younce, 2013, American journal of physiology. Cell physiology.

Monocyte Chemoattractant Protein-1 Induces a Novel Transcription Factor That Causes Cardiac Myocyte Apoptosis and Ventricular Dysfunction

Mahua Choudhury, S. Graham, P. Kolattukudy, 2006, Circulation research.

MCP-1 (Monocyte Chemotactic Protein-1)-induced Protein, a Recently Identified Zinc Finger Protein, Induces Adipogenesis in 3T3-L1 Pre-adipocytes without Peroxisome Proliferator-activated Receptor γ*

P. Kolattukudy, A. Azfer, Craig W. Younce, 2009, The Journal of Biological Chemistry.

Hyperglycaemia-induced cardiomyocyte death is mediated via MCP-1 production and induction of a novel zinc-finger protein MCPIP.

P. Kolattukudy, Kangkai Wang, Craig W. Younce, 2010, Cardiovascular research.

MCP-1 causes cardiomyoblast death via autophagy resulting from ER stress caused by oxidative stress generated by inducing a novel zinc-finger protein, MCPIP.

P. Kolattukudy, Craig W. Younce, 2010, The Biochemical journal.

Monocyte Chemoattractant Protein-1 Induces a Novel Transcription Factor That Causes Cardiac Myocyte Apoptosis and Ventricular Dysfunction

S. Graham, P. Kolattukudy, P. Binkley, 2006, Circulation research.