Yao-Chih Yang

Diallyl trisulfide protects against high glucose-induced cardiac apoptosis by stimulating the production of cystathionine gamma-lyase-derived hydrogen sulfide.

W. Kuo, Chih-Yang Huang, S. Wen, 2015, International journal of cardiology.

Nrf2 Activation as a Protective Feedback to Limit Cell Death in High Glucose‐Exposed Cardiomyocytes

W. Kuo, V. Viswanadha, Chih-Yang Huang, 2017, Journal of cellular biochemistry.

Pkcδ Activation is Involved in ROS-Mediated Mitochondrial Dysfunction and Apoptosis in Cardiomyocytes Exposed to Advanced Glycation End Products (Ages)

W. Kuo, Chih-Yang Huang, R. Aneja, 2018, Aging and disease.

ROS‐ and HIF1α‐dependent IGFBP3 upregulation blocks IGF1 survival signaling and thereby mediates high‐glucose‐induced cardiomyocyte apoptosis

W. Kuo, Chih-Yang Huang, S. Wen, 2019, Journal of cellular physiology.

Diallyl trisulfide suppresses doxorubicin‐induced cardiomyocyte apoptosis by inhibiting MAPK/NF‐κB signaling through attenuation of ROS generation

W. Kuo, P. Pai, Chih-Yang Huang, 2018, Environmental toxicology.

Lactate dehydrogenase downregulation mediates the inhibitory effect of diallyl trisulfide on proliferation, metastasis, and invasion in triple‐negative breast cancer

K. Ting, W. Kuo, V. Viswanadha, 2017, Environmental toxicology.