Keisuke Ishiwata

Genetic features of precursor B‐cell phenotype Burkitt leukemia with IGH‐ MYC rearrangement

Motohiro Kato, A. Sato-Otsubo, S. Ito, 2021, Cancer reports.

NUDT15 variants confer high incidence of second malignancies in children with acute lymphoblastic leukemia

K. Okamura, S. Ogawa, Motohiro Kato, 2021, Blood advances.

Rare variant of the epigenetic regulator SMCHD1 in a patient with pituitary hormone deficiency

K. Nagao, C. Obuse, T. Ogata, 2020, Scientific Reports.

Quantitative assessment of copy number alterations by liquid biopsy for neuroblastoma

Motohiro Kato, J. Takita, A. Sato-Otsubo, 2022, Genes, chromosomes & cancer.

Characteristics of genetic alterations of peripheral T‐cell lymphoma in childhood including identification of novel fusion genes: the Japan Children’s Cancer Group (JCCG)

K. Okamura, Y. Hayashi, Hiroko Ogata-Kawata, 2021, British journal of haematology.

Pseudoautosomal gene SHOX exhibits sex-biased random monoallelic expression and contributes to sex difference in height

K. Okamura, T. Ogata, A. Umezawa, 2021, bioRxiv.

Familial Pseudohypoparathyroidism Type IB Associated with an SVA Retrotransposon Insertion in the GNAS Locus

T. Ogata, H. Saitsu, K. Nakabayashi, 2022, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.

Rare variant of the epigenetic regulator SMCHD1 in a patient with pituitary hormone deficiency

K. Nagao, C. Obuse, T. Ogata, 2020, Scientific Reports.

A capture methyl-seq protocol with improved efficiency and cost-effectiveness using pre-pooling and enzymatic conversion

K. Nakabayashi, Y. Kasuga, Keisuke Ishiwata, 2023, BMC Research Notes.

Prevalence of pathogenic variants in cancer‐predisposing genes in second cancer after childhood solid cancers

K. Okamura, S. Ogawa, M. Takagi, 2023, Cancer medicine.

Variations in gender identity and sexual orientation of university students

Hiroko Ogata-Kawata, K. Nakabayashi, M. Fukami, 2023, Sexual medicine.

Development of sexual dimorphism of skeletal muscles through the adrenal cortex, caused by androgen-induced global gene suppression.

K. Nakabayashi, Shogo Yanai, Keisuke Ishiwata, 2024, Cell reports.