L. S. Ripley

发表

L. S. Ripley, 1988, Journal of molecular biology.

Hotspot sites for acridine-induced frameshift mutations in bacteriophage T4 correspond to sites of action of the T4 type II topoisomerase.

D. DeMarini, K. Kreuzer, J. deBoer, 1988, Journal of molecular biology.

Heat mutagenesis in bacteriophage T4: the transversion pathway.

J. Drake, P. Bingham, R. H. Baltz, 1976, Proceedings of the National Academy of Sciences of the United States of America.

Thymineless mutagenesis in bacteriophage T4.

J. Drake, M. Smith, L. S. Ripley, 1973, Genetics.

The spectrum of acridine resistant mutants of bacteriophage T4 reveals cryptic effects of the tsL141 DNA polymerase allele on spontaneous mutagenesis.

L. S. Ripley, F. J. Wang, 1998, Genetics.

Mutagenic specificity of nitrosomethylurea in bacteriophage T4.

L. S. Ripley, 1981, Mutation research.

Transversion mutagenesis in bacteriophage T4

L. S. Ripley, 1975, Molecular and General Genetics MGG.

Model for the participation of quasi-palindromic DNA sequences in frameshift mutation.

Taylor, L. S. Ripley, A., 1982, Proceedings of the National Academy of Sciences of the United States of America.

A proflavin-induced frameshift hotspot in the thymidylate synthase gene of bacteriophage T4.

D. Hall, M. D. Brown, L. S. Ripley, 1993, Mutation research.

An in vitro approach to identifying specificity determinants of mutagenesis mediated by DNA misalignments.

C. Papanicolaou, L. S. Ripley, 1991, Journal of molecular biology.

Frameshift mutation: determinants of specificity.

L. S. Ripley, L S Ripley, 1990, Annual review of genetics.

Structural intermediates of deletion mutagenesis: a role for palindromic DNA.

B. Glickman, L. S. Ripley, 1984, Proceedings of the National Academy of Sciences of the United States of America.

Spectrum of spontaneous frameshift mutations. Sequences of bacteriophage T4 rII gene frameshifts.

A. Clark, J. deBoer, L. S. Ripley, 1986, Journal of molecular biology.

Frameshift mutations produced by proflavin in bacteriophage T4: specificity within a hotspot.

A. Clark, L. S. Ripley, 1986, Proceedings of the National Academy of Sciences of the United States of America.

The specificity of infidelity of DNA polymerase.

L. S. Ripley, 1983, Basic life sciences.

The specificity of topoisomerase-mediated DNA cleavage defines acridine-induced frameshift specificity within a hotspot in bacteriophage T4.

K. Kreuzer, M. Masurekar, L. S. Ripley, 1991, Genetics.

Bacteriophage T4 particles are refractory to bisulfite mutagenesis.

J. Drake, L. S. Ripley, 1984, Mutation research.

Concerted Mutagenesis: Its Potential Impact on Interpretation of Evolutionary Relationships

L. S. Ripley, 1991 .

Primer-template misalignments during leading strand DNA synthesis account for the most frequent spontaneous mutations in a quasipalindromic region in Escherichia coli.

R. Sinden, W. A. Rosche, L. S. Ripley, 1998, Journal of molecular biology.

Demonstration of the production of frameshift and base-substitution mutations by quasipalindromic DNA sequences.

J. D. de Boer, L. S. Ripley, 1984, Proceedings of the National Academy of Sciences of the United States of America.

Deletion and duplication sequences induced in CHO cells by teniposide (VM-26), a topoisomerase II targeting drug, can be explained by the processing of DNA nicks produced by the drug-topoisomerase interaction.

L. S. Ripley, 1994, Mutation research.

A genetic assay for transversion mutations in bacteriophage T4

J. Drake, L. S. Ripley, 2006, Molecular and General Genetics MGG.

An in vitro assay for frameshift mutations: hotspots for deletions of 1 bp by Klenow-fragment polymerase share a consensus DNA sequence.

J. D. de Boer, L. S. Ripley, 1988, Genetics.

Polymerase infidelity and frameshift mutation.

N. Shoemaker, L. S. Ripley, 1982, Basic life sciences.

A structural model for sequence-specific proflavin-DNA interactions during in vitro frameshift mutagenesis.

H. Berman, L. Joshua-Tor, J. Sussman, 1992, Journal of biomolecular structure & dynamics.

Unique self-complementarity of palindromic sequences provides DNA structural intermediates for mutation.

B. Glickman, L. Ripley, L. S. Ripley, 1983, Cold Spring Harbor symposia on quantitative biology.

Polymerase-specific differences in the DNA intermediates of frameshift mutagenesis. In vitro synthesis errors of Escherichia coli DNA polymerase I and its large fragment derivative.

C. Papanicolaou, L. Ripley, L. S. Ripley, 1989, Journal of molecular biology.

A major role for bacteriophage T4 DNA polymerase in frameshift mutagenesis.

N. Shoemaker, L. S. Ripley, 1983, Genetics.

Mutator versus antimutator activity of a T4 DNA polymerase mutant distinguishes two different frameshifting mechanisms

B. Glickman, N. Shoemaker, L. S. Ripley, 2004, Molecular and General Genetics MGG.

Effects of proflavin and photoactivated proflavin on the template function of single-stranded DNA.

G. G. Revich, L. S. Ripley, 1990, Journal of molecular biology.

DNA sequence effects on single base deletions arising during DNA polymerization in vitro by Escherichia coli Klenow fragment polymerase.

L. S. Ripley, F. J. Wang, 1994, Genetics.

DNA nick processing by exonuclease and polymerase activities of bacteriophage T4 DNA polymerase accounts for acridine-induced mutation specificities in T4.

L. S. Ripley, V. Kaiser, 1995, Proceedings of the National Academy of Sciences of the United States of America.

Influence of diverse gene 43 DNA polymerases on the incorporation and replication in vivo of 2-aminopurine at A X T base-pairs in bacteriophage T4.

L. S. Ripley, 1981, Journal of molecular biology.