R. L. DuBois

Overlapping functions between XLF repair protein and 53BP1 DNA damage response factor in end joining and lymphocyte development

S. Zha, Wenxia Jiang, Z. Wolner, 2012, Proceedings of the National Academy of Sciences.

ATM Damage Response and XLF Repair Factor are Functionally Redundant In Joining DNA Breaks

F. Alt, C. Boboila, S. Zha, 2010, Nature.

Early B-cell-specific inactivation of ATM synergizes with ectopic CyclinD1 expression to promote pre-germinal center B-cell lymphomas in mice

S. Zha, G. Bhagat, E. Hobeika, 2015, Leukemia.

Kinase-dead ATM protein causes genomic instability and early embryonic lethality in mice

S. Zha, C. Bakkenist, Chyuan-Sheng Lin, 2012, The Journal of cell biology.

Ataxia telangiectasia-mutated protein and DNA-dependent protein kinase have complementary V(D)J recombination functions

F. Alt, S. Zha, Y. Fujiwara, 2011, Proceedings of the National Academy of Sciences.

Differential phosphorylation of DNA-PKcs regulates the interplay between end-processing and end-ligation during nonhomologous end-joining.

S. Nakajima, S. Zha, Xiaochun Yu, 2015, Molecular cell.

Differential Phosphorylatio n of DNA-PKcs Regulates the Interplay between End-Processing and End-Ligation during Nonhomologous End-Joining Graphical

S. Nakajima, S. Zha, Xiaochun Yu, 2015 .

Aberrant TCRδ rearrangement underlies the T-cell lymphocytopenia and t(12;14) translocation associated with ATM deficiency.

F. Alt, S. Zha, M. Gostissa, 2015, Blood.