Stephen J. Miller

发表

Random monoallelic expression of the imprinted IGF2 and H19 genes in the absence of discriminative parental marks

R. Ohlsson, H. Cui, G. Adam, 1999, Development Genes and Evolution.

Paternally derived H19 is differentially expressed in malignant and nonmalignant trophoblast.

R. Ohlsson, C. Walsh, S. Miller, 1995, Cancer research.

An Inr-containing sequence flanking the TATA box of the human c-sis (PDGF-B) proto-oncogene promoter functions in cis as a co-activator for its intronic enhancer.

R. Ohlsson, G. Franklin, G. Adam, 1995, Oncogene.

Genetic imprinting of IGF2/H19 in Normal, Hyperplastic and Neoplastic Cells

T. Ekström, R. Ohlsson, C. Walsh, 1996 .

A novel type of regulatory element is required for promoter-specific activity of the PDGF-B intronic enhancer region.

C. Walsh, G. Franklin, G. Adam, 1998, Growth factors.

The Sequential Activation and Repression of the Human PDGF-B Gene During Chronic Hypoxia Reveals Antagonistic Roles for the Depletion of Oxygen and Glucose

G. Franklin, A. Wilcock, S. Miller, 2001, Growth Factors.

Allele-specific in situ hybridization (ASISH) analysis: a novel technique which resolves differential allelic usage of H19 within the same cell lineage during human placental development.

R. Ohlsson, G. Adam, S. Miller, 1996, Development.

The paternal allele of the H19 gene is progressively silenced during early mouse development: the acetylation status of histones may be involved in the generation of variegated expression patterns.

R. Ohlsson, T. Olsson, S. Miller, 1998, Development.