E. Heidenreich

发表

Epistatic participation of REV1 and REV3 in the formation of UV-induced frameshift mutations in cell cycle-arrested yeast cells.

F. Steinboeck, E. Heidenreich, H. Eisler, 2006, Mutation research.

The sequence spectrum of frameshift reversions obtained with a novel adaptive mutation assay in Saccharomyces cerevisiae

F. Steinboeck, E. Heidenreich, 2016, Data in brief.

Replication-dependent and selection-induced mutations in respiration-competent and respiration-deficient strains of Saccharomyces cerevisiae

U. Wintersberger, E. Heidenreich, 1998, Molecular and General Genetics MGG.

Starvation for a specific amino acid induces high frequencies of rho− mutants in Saccharomyces cerevisiae

U. Wintersberger, E. Heidenreich, 1997, Current Genetics.

Starvation for an essential amino acid induces apoptosis and oxidative stress in yeast.

K. Fröhlich, E. Heidenreich, H. Eisler, 2004, Experimental cell research.

Non‐homologous end joining as an important mutagenic process in cell cycle‐arrested cells

Erich Heidenreich, Rene Novotny, Bernd Kneidinger, 2003, The EMBO journal.

The nuclear actin‐related protein Act3p/Arp4p of Saccharomyces cerevisiae is involved in transcription regulation of stress genes

K. Kuchler, I. Görzer, C. Schüller, 2003, Molecular microbiology.

Polymerase zeta dependency of increased adaptive mutation frequencies in nucleotide excision repair-deficient yeast strains.

E. Heidenreich, H. Eisler, V. Holzmann, 2004, DNA repair.

Novel regulatory properties of Saccharomyces cerevisiae Arp4.

A. Kaufmann, F. Steinboeck, E. Heidenreich, 2006, Journal of biochemistry.

The nuclear actin-related protein of Saccharomyces cerevisiae, Arp4, directly interacts with the histone acetyltransferase Esa1p.

A. Kaufmann, F. Steinboeck, E. Heidenreich, 2007, Journal of biochemistry.

A mutation-promotive role of nucleotide excision repair in cell cycle-arrested cell populations following UV irradiation.

F. Steinboeck, E. Heidenreich, H. Eisler, 2010, DNA repair.

Adaptive Mutation in Saccharomyces cerevisiae

E. Heidenreich, 2007, Critical reviews in biochemistry and molecular biology.

Adaptive reversions of a frameshift mutation in arrested Saccharomyces cerevisiae cells by simple deletions in mononucleotide repeats.

U. Wintersberger, E. Heidenreich, 2001, Mutation research.

Glucose starvation as a selective tool for the study of adaptive mutations in Saccharomyces cerevisiae.

F. Steinboeck, E. Heidenreich, 2017, Journal of microbiological methods.

The relevance of oxidative stress and cytotoxic DNA lesions for spontaneous mutagenesis in non-replicating yeast cells.

M. Hubmann, F. Steinboeck, E. Heidenreich, 2010, Mutation research.

Non-homologous end joining dependency of gamma-irradiation-induced adaptive frameshift mutation formation in cell cycle-arrested yeast cells.

E. Heidenreich, H. Eisler, Herfried Eisler, 2004, Mutation research.

A mutation-promotive role of nucleotide excision repair in cell cycle-arrested cell populations following UV irradiation.

F. Steinboeck, E. Heidenreich, Theresia Lengheimer, 2010, DNA repair.