R. J. Mann

Transgenic mice with chronically elevated luteinizing hormone are infertile due to anovulation, defects in uterine receptivity, and midgestation pregnancy failure.

R. Keri, J. Nilson, R. J. Mann, 1999, Endocrinology.

Chronically elevated luteinizing hormone depletes primordial follicles in the mouse ovary.

J. Flaws, A. Hirshfield, R. Abbud, 1997, Biology of reproduction.

Genetic and functional analyses of FH mutations in multiple cutaneous and uterine leiomyomatosis, hereditary leiomyomatosis and renal cancer, and fumarate hydratase deficiency.

N P Burrows, M Keefe, A. Rowan, 2003, Human molecular genetics.

Consequences of elevated luteinizing hormone on diverse physiological systems: use of the LHbetaCTP transgenic mouse as a model of ovarian hyperstimulation-induced pathophysiology.

R. Keri, J. Nilson, R. J. Mann, 2003, Recent progress in hormone research.

A G542X cystic fibrosis mouse model for examining nonsense mutation directed therapies

M. Drumm, C. Hodges, C. Cotton, 2018, PloS one.