David W. Van Norstrand

Overrepresentation of the proarrhythmic, sudden death predisposing sodium channel polymorphism S1103Y in a population-based cohort of African-American sudden infant death syndrome.

D. Tester, M. Ackerman, David W. Van Norstrand, 2008, Heart rhythm.

Light scattering measurement and Avrami analysis of the lamellar to inverse hexagonal phase transition kinetics of the lipid DEPE.

P. E. Harper, P. Cook, David W. Van Norstrand, 2012, Chemistry and physics of lipids.

&agr;1-Syntrophin Mutations Identified in Sudden Infant Death Syndrome Cause an Increase in Late Cardiac Sodium Current

C. January, D. Tester, M. Ackerman, 2009, Circulation. Arrhythmia and electrophysiology.

Molecular and Functional Characterization of Novel Glycerol-3-Phosphate Dehydrogenase 1–Like Gene (GPD1-L) Mutations in Sudden Infant Death Syndrome

D. Tester, M. Ackerman, C. Valdivia, 2007, Circulation.

Genomic risk factors in sudden infant death syndrome

M. Ackerman, David W. Van Norstrand, M. Ackerman, 2010, Genome Medicine.

Sudden infant death syndrome: do ion channels play a role?

M. Ackerman, David W. Van Norstrand, M. Ackerman, 2009, Heart rhythm.

Connexin43 Mutation Causes Heterogeneous Gap Junction Loss and Sudden Infant Death

D. Tester, M. Ackerman, J. Saffitz, 2012, Circulation.

Sudden infant death syndrome-associated mutations in the sodium channel beta subunits.

D. Tester, M. Ackerman, C. Valdivia, 2010, Heart rhythm.