J. Leith

Assessing human alloimmunization as a strategy for inducing HIV type 1 neutralizing anti-HLA responses.

M. Luscher, K. MacDonald, K. Rosenthal, 2003, AIDS research and human retroviruses.

CD8+ T-cell-mediated suppression of HIV-1 long terminal repeat-driven gene expression is not modulated by the CC chemokines RANTES, macrophage inflammatory protein (MIP)-1 a and MIP-1 b

K. Rosenthal, C. Richards, K. Copeland, 2018 .

CD8+ T‐cell‐mediated suppression of HIV‐1 long terminal repeat‐driven gene expression is not modulated by the CC chemokines RANTES, macrophage inflammatory protein (MIP)‐1α and MIP‐1β

K. Rosenthal, C. Richards, K. Copeland, 1997 .

CD8+ T cell‐mediated suppression of HIV long terminal repeat‐driven gene expression is not associated with improved clinical status

K. Rosenthal, K. Copeland, F. Smaill, 1997, AIDS.

CD8+ T cell supernatants of HIV type 1-infected individuals have opposite effects on long terminal repeat-mediated transcription in T cells and monocytes.

K. Rosenthal, K. Copeland, J. Leith, 1997, AIDS research and human retroviruses.

T cell-derived suppressive activity: evidence of autocrine noncytolytic control of HIV type 1 transcription and replication.

K. Rosenthal, D. Bienzle, C. Richards, 1999, AIDS research and human retroviruses.

Oncostatin M stimulates monocyte chemoattractant protein-1- and interleukin-1-induced matrix metalloproteinase-1 production by human synovial fibroblasts in vitro.

C. Richards, C. Langdon, J. Leith, 1997, Arthritis and rheumatism.

CD8+ T-cell-mediated suppression of HIV-1 long terminal repeat-driven gene expression is not modulated by the CC chemokines RANTES, macrophage inflammatory protein (MIP)-1 alpha and MIP-1 beta.

K. Rosenthal, C. Richards, K. Copeland, 1997, AIDS.