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Replication inhibitors modulate instability of an expanded trinucleotide repeat at the myotonic dystrophy type 1 disease locus in human cells.

D. Chitayat, C. E. Pearson, Julien L. Marcadier, 2003, American journal of human genetics.

Genomic context drives SCA7 CAG repeat instability, while expressed SCA7 cDNAs are intergenerationally and somatically stable in transgenic mice.

C. Ware, Ying-Hui Fu, C. E. Pearson, 2003, Human molecular genetics.

Antagonistic roles of canonical and alternative RPA in tandem CAG repeat diseases

F. Boisvert, B. Davidson, A. L. Deshmukh, 2022, bioRxiv.

CTCF cis-Regulates Trinucleotide Repeat Instability in an Epigenetic Manner: A Novel Basis for Mutational Hot Spot Determination

Stephen J. Tapscott, S. Tapscott, G. Filippova, 2008, PLoS genetics.

Genetic Complexity of Cellulose Synthase A Gene Function in Arabidopsis Embryogenesis1

Gerhard K. H. Przemeck, D. Inzé, N. Terryn, 2002, Plant Physiology.

CHAPTER 41 – Error-Prone Repair of Slipped (CTG)·(CAG) Repeats and Disease-Associated Expansions

Michelle R. Leonard, C. E. Pearson, Julien L. Marcadier, 2006 .

Slipped (CTG)•(CAG) repeats can be correctly repaired, escape repair or undergo error-prone repair

Michelle R. Leonard, C. E. Pearson, S. Montgomery, 2005, Nature Structural &Molecular Biology.