Shannon Zelina

发表

Mechanisms by Which the G333D Mutation in Human Immunodeficiency Virus Type 1 Reverse Transcriptase Facilitates Dual Resistance to Zidovudine and Lamivudine

J. Mellors, N. Sluis-Cremer, J. Radzio, 2007, Antimicrobial Agents and Chemotherapy.

The Human Immunodeficiency Virus Type 1 Nonnucleoside Reverse Transcriptase Inhibitor Resistance Mutation I132M Confers Hypersensitivity to Nucleoside Analogs

B. Herman, N. Sluis-Cremer, Z. Ambrose, 2009, Journal of Virology.

Molecular mechanisms of bidirectional antagonism between K65R and thymidine analog mutations in HIV-1 reverse transcriptase

J. Mellors, N. Sluis-Cremer, U. Parikh, 2007, AIDS.

Molecular Mechanism by Which the K70E Mutation in Human Immunodeficiency Virus Type 1 Reverse Transcriptase Confers Resistance to Nucleoside Reverse Transcriptase Inhibitors

J. Mellors, N. Sluis-Cremer, U. Parikh, 2006, Antimicrobial Agents and Chemotherapy.

Regulatory Issues in the Therapeutic Use of Stem Cells

B. Deasy, Jordan E. Anderson, Shannon Zelina, 2013 .

Impact of residues in the nonnucleoside reverse transcriptase inhibitor binding pocket on HIV-1 reverse transcriptase heterodimer stability.

N. Sluis-Cremer, G. Tachedjian, A. Figueiredo, 2008, Current HIV research.