S. Wagner

发表

Cytoskeleton and epithelial polarity.

D. Drenckhahn, R. Koob, T. Jöns, 1993, Renal physiology and biochemistry.

Intercalated cells as a probable source for the development of renal oncocytoma

S. Störkel, P. Steart, W. Thoenes, 1988, Virchows Archiv. B, Cell pathology including molecular pathology.

The dominant chloride channel mutant G200R causing fluctuating myotonia: Clinical findings, electrophysiology, and channel pathology

F. Lehmann-Horn, F. Deymeer, C. Özdemir, 1998, Muscle & nerve.

Probing the major skeletal muscle chloride channel with Zn2+ and other sulfhydryl-reactive compounds

R. Rüdel, Alfred L. George Jr., S. Wagner, 1996, Pflügers Archiv.

K(+)‐aggravated myotonia: destabilization of the inactivated state of the human muscle Na+ channel by the V1589M mutation.

H. Lerche, F. Lehmann-Horn, C. Fahlke, 1994, The Journal of physiology.

Anion exchanger 1 in human kidney and oncocytoma differs from erythroid AE1 in its NH2 terminus.

D. Drenckhahn, H. Appelhans, A. Kollert-Jöns, 1993, The American journal of physiology.

Band 3 is the basolateral anion exchanger of dark epithelial cells of turtle urinary bladder.

D. Drenckhahn, P. Schaaf, S. Wagner, 1987, The American journal of physiology.

Different effects on gating of three myotonia‐causing mutations in the inactivation gate of the human muscle sodium channel.

H. Lerche, F. Lehmann-Horn, C. Fahlke, 1995, The Journal of physiology.

Immunochemical characterization of a band 3-like anion exchanger in collecting duct of human kidney.

R. Lietzke, D. Drenckhahn, R. Koob, 1987, The American journal of physiology.

A novel sodium channel mutation causing a hyperkalemic paralytic and paramyotonic syndrome with variable clinical expressivity

H. Lerche, F. Lehmann-Horn, N. Mitrović, 1997, Neurology.

Role of Na+,K+-ATPase and anion exchanger 1 (band 3) as binding sites for the cytoskeleton

D. Drenckhahn, R. Koob, T. Jöns, 1992 .