N. Hilliard

Molecular Determinants of Frequency Dependence and Ca 2 (cid:1) Potentiation of Verapamil Block in the Pore Region of Ca v 1.2

N. Dilmac, G. Hockerman, N. Hilliard, 2004 .

Molecular Determinants of Frequency Dependence and Ca2+ Potentiation of Verapamil Block in the Pore Region of Cav1.2

N. Dilmac, G. Hockerman, N. Hilliard, 2004, Molecular Pharmacology.

Differential Modulation of Cav1.2 and Cav1.3-Mediated Glucose-Stimulated Insulin Secretion by cAMP in INS-1 Cells: Distinct Roles for Exchange Protein Directly Activated by cAMP 2 (Epac2) and Protein Kinase A

Guohong Liu, G. Hockerman, S. M. Jacobo, 2006, Journal of Pharmacology and Experimental Therapeutics.

Molecular determinants of Ca2+ potentiation of diltiazem block and Ca2+-dependent inactivation in the pore region of cav1.2.

N. Dilmac, G. Hockerman, N. Hilliard, 2003, Molecular pharmacology.

Molecular Determinants of Ca 2 Potentiation of Diltiazem Block and Ca 2-Dependent Inactivation in the Pore Region of Cav 1 . 2

N. Dilmac, G. Hockerman, N. Hilliard, 2003 .

Cav1.3 Is Preferentially Coupled to Glucose-Stimulated Insulin Secretion in the Pancreatic β-Cell Line INS-1

N. Dilmac, Guohong Liu, G. Hockerman, 2003, Journal of Pharmacology and Experimental Therapeutics.

Ca v 1.3 is preferentially coupled to glucose-stimulated insulin secretion in the pancreatic beta-cell line INS-1.

N. Dilmac, Guohong Liu, G. Hockerman, 2003, The Journal of pharmacology and experimental therapeutics.

Cav1.3 is preferentially coupled to glucose-induced [Ca2+]i oscillations in the pancreatic beta cell line INS-1.

Guohong Liu, G. Hockerman, N. Hilliard, 2004, Molecular pharmacology.

Adenoviral-mediated expression of dihydropyridine-insensitive L-type calcium channels in cardiac ventricular myocytes and fibroblasts.

J. Fuseler, Jining Zhang, John W Fuseler, 2007, European journal of pharmacology.