J. Nistal

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Sex-Speciﬁc Regulation of miR-29b in the Myocardium Under Pressure Overload is Associated with Di ﬀ erential Molecular, Structural and Functional Remodeling Patterns in Mice and Patients with Aortic Stenosis

A. Villar, D. Merino, R. Garc, 2020 .

Experimental modelling of cardiac pressure overload hypertrophy: Modified technique for precise, reproducible, safe and easy aortic arch banding-debanding in mice

M. Hurlé, J. Nistal, M. Llano, 2018, Scientific Reports.

Myocardial gene expression of microRNA-133a and myosin heavy and light chains, in conjunction with clinical parameters, predict regression of left ventricular hypertrophy after valve replacement in patients with aortic stenosis

M. Hurlé, J. Hurlé, M. Llano, 2011, Heart.

Myocardial and circulating levels of microRNA-21 reflect left ventricular fibrosis in aortic stenosis patients.

M. Hurlé, J. Nistal, M. Llano, 2013, International journal of cardiology.

Nitric oxide mediates aortic disease in mice deficient in the metalloprotease Adamts1 and in a mouse model of Marfan syndrome

J. Redondo, D. Milewicz, A. Evangelista, 2017, Nature Medicine.

Androgens Contribute to Sex Differences in Myocardial Remodeling under Pressure Overload by a Mechanism Involving TGF-β

M. Ares, M. Hurlé, J. Nistal, 2012, PloS one.

Elsevier Editorial System(tm) for International Journal of Cardiology Manuscript Draft Title: Myocardial and Circulating Levels of Microrna-21 Reflect Left Ventricular Fibrosis in Aortic Stenosis Patients International Journal of Cardiology Author Agreement Form Myocardial and Circulating Levels of

M. Hurlé, M. Llano, A. Villar, 2022 .

Gender differences of echocardiographic and gene expression patterns in human pressure overload left ventricular hypertrophy.

M. Hurlé, R. Merino, J. Nistal, 2009, Journal of molecular and cellular cardiology.

BMP7-based peptide agonists of BMPR1A protect the left ventricle against pathological remodeling induced by pressure overload.

J. Redondo, M. Hurlé, J. Nistal, 2022, Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie.

Extracellular heat shock protein 90 binding to TGFβ receptor I participates in TGFβ-mediated collagen production in myocardial fibroblasts.

A. Cortajarena, M. Hurlé, J. Nistal, 2016, Cellular signalling.

Myocardial gene expression of microRNA-133a and myosin heavy and light chains, in conjunction with clinical parameters, predict regression of left ventricular hypertrophy after valve replacement in patients with aortic stenosis

M. Hurlé, J. Hurlé, M. Llano, 2011, Heart.

BAMBI (BMP and activin membrane-bound inhibitor) protects the murine heart from pressure-overload biomechanical stress by restraining TGF-β signaling.

M. Hurlé, J. Hurlé, J. Nistal, 2013, Biochimica et biophysica acta.

SIRT3-mediated inhibition of FOS through histone H3 deacetylation prevents cardiac fibrosis and inflammation

F. Villarroya, I. Miguel-Escalada, F. Crispi, 2020, Signal Transduction and Targeted Therapy.

p-SMAD2/3 and DICER promote pre-miR-21 processing during pressure overload-associated myocardial remodeling.

C. Fernández-Hernando, Arantxa González, M. Hurlé, 2015, Biochimica et biophysica acta.

Plasma Levels of Transforming Growth Factor-β1 Reflect Left Ventricular Remodeling in Aortic Stenosis

M. Hurlé, J. Nistal, M. Llano, 2009, PloS one.