S. Seyen

Mutation location and IKs regulation in the arrhythmic risk of long QT syndrome type 1: the importance of the KCNQ1 S6 region.

P. Schwartz, P. Brink, P. Volders, 2021, European heart journal.

Long-QT mutation p.K557E-Kv7.1: dominant-negative suppression of IKs, but preserved cAMP-dependent up-regulation.

Viola Lentink, P. Volders, J. Heijman, 2014, Cardiovascular research.

Benfotiamine, a synthetic S-acyl thiamine derivative, has different mechanisms of action and a different pharmacological profile than lipid-soluble thiamine disulfide derivatives

Brigitte Evrard, L. Bettendorff, B. Evrard, 2008, BMC pharmacology.

Nfat and miR-25 cooperate to reactivate the transcription factor Hand2 in heart failure

S. Heymans, M. Gladka, P. Volders, 2013, Nature Cell Biology.

Dominant-Negative Control of cAMP-Dependent IKs Upregulation in Human Long-QT Syndrome Type 1

Viola Lentink, P. Volders, L. D. de Windt, 2012, Circulation research.

The usefulness of operant conditioning procedures to assess long-lasting deficits following transient focal ischemia in mice

A. Ferrara, E. Tirelli, J. Plumier, 2009, Behavioural Brain Research.

Benfotiamine, a synthetic S-acyl thiamine derivative, has different mechanisms of action and a different pharmacological profile than lipid-soluble thiamine disulfide derivatives

L. Bettendorff, B. Evrard, J. Plumier, 2008, BMC pharmacology.