Te Du

发表

PRESSURE-DRIVEN RELEASE OF VIRAL GENOME INTO A HOST NUCLEUS IS A 1 MECHANISM LEADING TO HERPES INFECTION 2 3 4 5

A. Evilevitch, A. Brandariz-Nuñez, Ting Liu, 2022 .

Induction of apoptosis accelerates reactivation of latent HSV-1 in ganglionic organ cultures and replication in cell cultures

B. Roizman, Guoying Zhou, Te Du, 2012, Proceedings of the National Academy of Sciences.

Disruption of HDAC/CoREST/REST repressor by dnREST reduces genome silencing and increases virulence of herpes simplex virus

B. Roizman, Guoying Zhou, Haidong Gu, 2010, Proceedings of the National Academy of Sciences.

Role of activating transcription factor 3 in the synthesis of latency-associated transcript and maintenance of herpes simplex virus 1 in latent state in ganglia

B. Roizman, M. Shu, G. Zhou, 2015, Proceedings of the National Academy of Sciences.

The Role of the CoREST/REST Repressor Complex in Herpes Simplex Virus 1 Productive Infection and in Latency

B. Roizman, Guoying Zhou, Te Du, 2013, Viruses.

HSV carrying WT REST establishes latency but reactivates only if the synthesis of REST is suppressed

B. Roizman, Guoying Zhou, Te Du, 2013, Proceedings of the National Academy of Sciences.

Checkpoints in productive and latent infections with herpes simplex virus 1: conceptualization of the issues

B. Roizman, Guoying Zhou, Te Du, 2011, Journal of NeuroVirology.

HSV-1 gene expression from reactivated ganglia is disordered and concurrent with suppression of latency-associated transcript and miRNAs

B. Roizman, Guoying Zhou, Te Du, 2011, Proceedings of the National Academy of Sciences.

miR-H28 and miR-H29 expressed late in productive infection are exported and restrict HSV-1 replication and spread in recipient cells

B. Roizman, Guoying Zhou, Xusha Zhou, 2016, Proceedings of the National Academy of Sciences.

Patterns of accumulation of miRNAs encoded by herpes simplex virus during productive infection, latency, and on reactivation

Bernard Roizman, Zhiyuan Han, B. Roizman, 2014, Proceedings of the National Academy of Sciences.

The Stability of Herpes Simplex Virus 1 ICP0 Early after Infection Is Defined by the RING Finger and the UL13 Protein Kinase

B. Roizman, Guoying Zhou, Zhi Zhu, 2014, Journal of Virology.

Cells infected with herpes simplex virus 1 export to uninfected cells exosomes containing STING, viral mRNAs, and microRNAs

B. Roizman, M. Kalamvoki, Te Du, 2014, Proceedings of the National Academy of Sciences.

Pressure-driven release of viral genome into a host nucleus is a mechanism leading to herpes infection

A. Evilevitch, A. Brandariz-Nuñez, Ting Liu, 2019, eLife.

Modulation of reactivation of latent herpes simplex virus 1 in ganglionic organ cultures by p300/CBP and STAT3

B. Roizman, Guoying Zhou, Te Du, 2013, Proceedings of the National Academy of Sciences.

miR-H28 and miR-H29 expressed late in productive infection are exported and restrict HSV-1 replication and spread in recipient cells

B. Roizman, Guoying Zhou, Xusha Zhou, 2016, Proceedings of the National Academy of Sciences.

Patterns of accumulation of miRNAs encoded by herpes simplex virus during productive infection, latency, and on reactivation

B. Roizman, Guoying Zhou, Zhiyuan Han, 2014, Proceedings of the National Academy of Sciences.