E. Frithz‐Lindsten

The chaperone‐like protein YerA of Yersinia pseudotuberculosis stabilizes YopE in the cytoplasm but is dispensible for targeting to the secretion loci

R. Rosqvist, A. Forsberg, L. Johansson, 1995, Molecular microbiology.

Exoenzyme T of Pseudomonas aeruginosa elicits cytotoxicity without interfering with Ras signal transduction

B. Hallberg, M. L. Henriksson, A. Forsberg, 2001, Cellular microbiology.

Intracellular targeting of exoenzyme S of Pseudomonas aeruginosa via type III‐dependent translocation induces phagocytosis resistance, cytotoxicity and disruption of actin microfilaments

R. Rosqvist, A. Forsberg, E. Frithz‐Lindsten, 1997, Molecular microbiology.

The V‐antigen of Yersinia is surface exposed before target cell contact and involved in virulence protein translocation

R. Titball, A. Holmström, H. Wolf‐Watz, 1999, Molecular microbiology.

Functional conservation of the effector protein translocators PopB/YopB and PopD/YopD of Pseudomonas aeruginosa and Yersinia pseudotuberculosis

M Soltani, A. Holmström, R. Rosqvist, 1998, Molecular microbiology.

Direct repeat‐mediated deletion of a type IV pilin gene results in major virulence attenuation of Francisella tularensis

Mats Forsman, Richard W Titball, Laila Noppa, 2006, Molecular microbiology.

Delineation and mutational analysis of the Yersinia pseudotuberculosis YopE domains which mediate translocation across bacterial and eukaryotic cellular membranes

H. Wolf‐Watz, K. Schesser, E Frithz-Lindsten, 1996, Journal of bacteriology.

Type IV pili are not specifically required for contact dependent translocation of exoenzymes by Pseudomonas aeruginosa.

S. Lory, M. Wolfgang, A. Forsberg, 2002, Microbial pathogenesis.