N. Thummala

A non-immunogenic adenoviral vector, coexpressing CTLA4Ig and bilirubin-uridine-diphosphoglucuronateglucuronosyltransferase permits long-term, repeatable transgene expression in the Gunn rat model of Crigler–Najjar syndrome

S. Lee, S. Ghosh, B. Reddy, 2002, Gene Therapy.

Insertion of the adenoviral E3 region into a recombinant viral vector prevents antiviral humoral and cellular immune responses and permits long-term gene expression.

G. Droguett, M. Horwitz, N. Chowdhury, 1997, Proceedings of the National Academy of Sciences of the United States of America.

Splice-site mutations: a novel genetic mechanism of Crigler-Najjar syndrome type 1.

P. Bosma, B. Deocharan, M. Sinaasappel, 1998, American journal of human genetics.