G. Tomer

The mutagenesis proteins UmuD' and UmuC prevent lethal frameshifts while increasing base substitution mutations.

Z. Livneh, N. Reuven, G. Tomer, 1998, Molecular cell.

Highly mutagenic replication by DNA polymerase V (UmuC) provides a mechanistic basis for SOS untargeted mutagenesis.

Z. Livneh, N. Reuven, Z Livneh, 2000, Proceedings of the National Academy of Sciences of the United States of America.

Parallel multiplicative target screening against divergent bacterial replicases: identification of specific inhibitors with broad spectrum potential.

Oliver J. Fackelmayer, P. Burgers, C. McHenry, 2010, Biochemistry.

Reconstitution of repair-gap UV mutagenesis with purified proteins from Escherichia coli: a role for DNA polymerases III and II.

Z. Livneh, M. O’Donnell, O. Cohen-Fix, 1996, Proceedings of the National Academy of Sciences of the United States of America.

Contribution of Human Mlh1 and Pms2 ATPase Activities to DNA Mismatch Repair*

R. Liskay, A. Buermeyer, Megan Nguyen, 2002, The Journal of Biological Chemistry.

Truncation of the C-terminus of human MLH1 blocks intracellular stabilization of PMS2 and disrupts DNA mismatch repair.

Xin Huo, A. Buermeyer, Megan Nguyen, 2006, DNA repair.

Analysis of unassisted translesion replication by the DNA polymerase III holoenzyme.

Z. Livneh, G. Tomer, 1999, Biochemistry.

The E705K mutation in hPMS2 exerts recessive, not dominant, effects on mismatch repair.

R. Liskay, N. Erdeniz, Natalia Sepúlveda, 2007, Cancer letters.

The beta subunit sliding DNA clamp is responsible for unassisted mutagenic translesion replication by DNA polymerase III holoenzyme.

Z. Livneh, N. Reuven, G. Tomer, 1998, Proceedings of the National Academy of Sciences of the United States of America.