K. Maloney

TEL-AML1 fusion identifies a subset of children with standard risk acute lymphoblastic leukemia who have an excellent prognosis when treated with therapy that includes a single delayed intensification

L. Mcgavran, L. Stork, K. Maloney, 1999, Leukemia.

Acquisition of p16(INK4A) and p15(INK4B) gene abnormalities between initial diagnosis and relapse in children with acute lymphoblastic leukemia.

S. Hunger, L. Mcgavran, K. Maloney, 1999, Blood.

Lack of ETV6 (TEL) gene rearrangements or p16INK4A/p15INK4B homozygous gene deletions in infant acute lymphoblastic leukemia

J. Rubnitz, D. Pullen, N. Hakami, 1997, Leukemia.

Acquisition of p16(INK4A) and p15(INK4B) gene abnormalities between initial diagnosis and relapse in children with acute lymphoblastic leukemia.

S. Hunger, L. Mcgavran, K. Maloney, 1999, Blood.

Different patterns of homozygous p16INK4A and p15INK4B deletions in childhood acute lymphoblastic leukemias containing distinct E2A translocations

L. Mcgavran, S. Hunger, K. Maloney, 1998, Leukemia.

Long-term results of treatment studies for childhood acute lymphoblastic leukemia: Pediatric Oncology Group studies from 1986–1994

J. Shuster, J. Pullen, S. Murphy, 2000, Leukemia.