F. Piattini

发表

Apolipoprotein E controls Dectin-1-dependent development of monocyte-derived alveolar macrophages upon pulmonary β-glucan-induced inflammatory adaptation

F. Ginhoux, A. Schlitzer, J. Ruland, 2022, bioRxiv.

KappaBle fluorescent reporter mice enable low-background single-cell detection of NF-κB transcriptional activity in vivo

T. Rülicke, W. Hardt, M. Kopf, 2022, Mucosal Immunology.

Monocyte-derived alveolar macrophages autonomously determine severe outcome of respiratory viral infection

H. Rehrauer, M. Kopf, Fengqi Li, 2022, Science Immunology.

A spontaneous leptin receptor point mutation causes obesity and differentially affects leptin signaling in hypothalamic nuclei resulting in metabolic dysfunctions distinct from db/db mice

M. Kopf, C. Schneider, J. Kisielow, 2019, Molecular metabolism.

Gene therapy of Csf2ra deficiency in mouse fetal monocyte precursors restores alveolar macrophage development and function

M. Kopf, C. Schneider, Fengqi Li, 2022, JCI insight.

Differential sensitivity of inflammatory macrophages and alternatively activated macrophages to ferroptosis

M. Kopf, C. Schneider, P. Bretscher, 2020, European journal of immunology.

The role of recruitment versus training in influenza-induced lasting changes to alveolar macrophage function

M. Guilliams, M. Kopf, C. Jakubzick, 2023, Nature Immunology.

Apolipoprotein E controls Dectin-1-dependent development of monocyte-derived alveolar macrophages upon pulmonary β-glucan-induced inflammatory adaptation.

F. Ginhoux, A. Schlitzer, J. Schulte-Schrepping, 2024, Nature immunology.