I. Klingel-schmitt

Evidence for an Upregulation of Acetylcholine Receptor Messenger Subunits Triggered by Antibody-Mediated Receptor Internalization in Human Myasthenia Gravis Muscles

V. Mouly, D. Beeson, S. Berrih-Aknin, 2000 .

Regulation of acetylcholine receptor gene expression in human myasthenia gravis muscles. Evidences for a compensatory mechanism triggered by receptor loss.

V. Mouly, D. Beeson, S. Berrih-Aknin, 1998, The Journal of clinical investigation.

Interleukin-6 overproduction by cultured thymic epithelial cells from patients with myasthenia gravis is potentially involved in thymic hyperplasia.

S. Berrih-Aknin, S. Cohen-Kaminsky, P. Galanaud, 1993, European cytokine network.

In vivo preferential usage of TCR V/gb8 in Torpedo acetylcholine receptor immune response in the murine experimental model of myasthenia gravis

C. Aimé-sempé, S. Berrih-Aknin, S. Cohen-Kaminsky, 1995, Journal of Neuroimmunology.

Sustained calcium signalling and caspase-3 activation involve NMDA receptors in thymocytes in contact with dendritic cells

G. Collingridge, S. Peineau, R. Liblau, 2011, Cell Death and Differentiation.

High IL‐6 Gene Expression and Production by Cultured Human Thymic Epithelial Cells from Patients with Myasthenia Gravis

S. Berrih-Aknin, S. Cohen-Kaminsky, P. Galanaud, 1993, Annals of the New York Academy of Sciences.

Circulating regulatory anti-T cell receptor antibodies in patients with myasthenia gravis.

Wei Zhang, Olivier Michel, Wei Zhang, 2003, The Journal of clinical investigation.

Rationale for a T Cell Receptor Peptide Therapy in Myasthenia Gravis

S. Berrih-Aknin, S. Cohen-Kaminsky, S. Caillat-Zucman, 2003, Annals of the New York Academy of Sciences.