RenateB. Alvarez

发表

Short Communication Light and Electron Microscopic Localization of ,B-amyloid Protein in Muscle Biopsies of Patients with Inclusion-body Myositis

W. Engel, V. Askanas, RenateB. Alvarez, 2007 .

Fourteen Newly Recognized Proteins at the Human Neuromuscular Junctions‐and Their Nonjunctional Accumulation in Inclusion‐Body Myositisa a

W. Engel, V. Askanas, V Askanas, 1998, Annals of the New York Academy of Sciences.

De novo neuromuscular junction formation on human muscle fibres cultured in monolayer and innervated by foetal rat spinal cord: Ultrastructural and ultrastructural-cytochemical studies

W. Engel, Takayoshi Kobayashi, A. Martinuzzi, 1987, Journal of neurocytology.

Difference in Expression of Phosphorylated Tau Epitopes between Sporadic Inclusion‐body Myositis and Hereditary Inclusion‐body Myopathies

W. Engel, M. Mirabella, V. Askanas, 1996, Journal of neuropathology and experimental neurology.

βAPP gene transfer into cultured human muscle induces inclusion‐body myositis aspects

W. Engel, V. Askanas, V Askanas, 1997 .

β-amyloid protein immunoreactivity in muscle of patients with inclusion-body myositis

W. Engel, G. Glenner, V. Askanas, 1992, The Lancet.

Increase of nitric oxide synthases and nitrotyrosine in inclusion‐body myositis

W. Engel, V. Askanas, V Askanas, 1996, Neuroreport.

Use of anti‐neurofilament antibody to identify paired‐helical filaments in inclusion‐body myositis

W. Engel, M. Mirabella, V. Askanas, 1996, Annals of neurology.

Immunocytochemical localization of ubiquitin in inclusion body myositis allows its light‐microscopic distinction from polymyositis

W. Engel, V. Askanas, V Askanas, 1992, Neurology.

Prion protein is abnormally accumulated in inclusion‐body myositis

W. Engel, F. Tomé, V. Askanas, 1993, Neuroreport.

Apolipoprotein E immunoreactive deposits in inclusion-body muscle diseases

W. Engel, K. Weisgraber, M. Mirabella, 1994, The Lancet.

beta-Amyloid precursor protein mRNA is increased in inclusion-body myositis muscle.

W. Engel, V. Askanas, V Askanas, 1993, Neuroreport.

Twisted tubulofilaments of inclusion body myositis muscle resemble paired helical filaments of Alzheimer brain and contain hyperphosphorylated tau.

D. Selkoe, W. Engel, V. Askanas, 1994, The American journal of pathology.

Transfer of beta-amyloid precursor protein gene using adenovirus vector causes mitochondrial abnormalities in cultured normal human muscle.

W. Engel, V. Askanas, V Askanas, 1996, Proceedings of the National Academy of Sciences of the United States of America.

Light and electron microscopic immunolocalization of presenilin 1 in abnormal muscle fibers of patients with sporadic inclusion-body myositis and autosomal-recessive inclusion-body myopathy.

W. Engel, T. Wisniewski, V. Askanas, 1998, The American journal of pathology.